Dieroff H G
Audiology. 1976;15(2):152-62. doi: 10.3109/00206097609071771.
Several audiometric tests were made of 85 noise workers exposed to an intensive impulse noise during pressing and punching. These tests aimed at finding out noise-induced central hearing damages (directional audiometry, dichotic speech discrimination test according to Feldmann and checking of the induction of the impedance jump). In addition, a clinical EEG was taken from each subject. Having obtained the necessary norms with series of tests in subjects with normal hearing abilities for their respective ages, the following results have been obtained: (1) directional hearing has hardly been affected by noise-induced hearing damage. It behaves like age changes in the group compared with it; (2) the dichotic speech discrimination test shows increasingly pathological values with progressive noise-induced hearing damage; this was already obvious in the age group 41-50. The impedance jump is less and less produced with progressive noise-induced hearing damage and finally disappears at a noise intensity of up to 110 dB. Because of these findings a progressive degeneration of the spiral ganglion and hearing nerve in noise-induced hearing damage is discussed as a cause of a deteriorated speech discrimination and the absence of the impedance jump. The EEG findings show changes in the alpha-rhythm in about 50% of the cases at an exposure time of 10 years onwards which may be possibly due to damage to the reticular formation, once increasing noise-induced hearing damage no longer exists.
对85名在冲压过程中暴露于强烈脉冲噪声的噪声作业工人进行了多项听力测试。这些测试旨在找出噪声引起的中枢性听力损伤(定向听力测定、根据费尔德曼进行的双耳语音辨别测试以及检查阻抗跃变的诱发情况)。此外,还对每个受试者进行了临床脑电图检查。在对听力正常的各年龄段受试者进行一系列测试并获得必要的正常标准后,得到了以下结果:(1)定向听力几乎未受噪声性听力损伤的影响。与对照组相比,其变化类似于年龄变化;(2)双耳语音辨别测试显示,随着噪声性听力损伤的加重,病理值越来越高;这在41 - 50岁年龄组中已经很明显。随着噪声性听力损伤的加重,阻抗跃变越来越少出现,最终在高达110分贝的噪声强度下消失。基于这些发现,有人讨论了噪声性听力损伤中螺旋神经节和听神经的渐进性退化是语音辨别能力下降和阻抗跃变消失的原因。脑电图结果显示,在暴露10年及以后,约50%的病例出现了α波节律变化,一旦噪声性听力损伤不再加重,这可能是由于网状结构受损所致。