Glasenapp G B
Laryngol Rhinol Otol (Stuttg). 1975 Jun;54(6):458-61.
The striking frequency of tracheal stenoses following laryngectomy gave reason for considerations on the typical causes. In our case-materials 9 tracheal stenoses were observed following 229 laryngectomies during the period from 1952 to 1972. Two of these cases represent typical stenoses produced by pressure. Within a short period of time, from 2 days up to 6 months after operation the 7 remaining cases represented marked tracheal stenoses with the mucosae showing no signes of irritation. These stenoses had been registered since 1965 only, i.e. after the introduction of the general application of insufflation anesthesia in every case of laryngectomy at our clinic. These observations gave reason for supposing that the procedure of anesthesia had to be seen as responsible for the development of tracheal stenoses. Under unfavourable circumstances during operation, even if the tube is inserted for a short period of time the anemising pressure effect of the cuff of the tube might - beside other wellknown factors - produce damages on the tracheal wall.
喉切除术后气管狭窄的发生率惊人，这促使人们对其典型病因进行思考。在我们的病例资料中，1952年至1972年间，229例喉切除术后观察到9例气管狭窄。其中2例为典型的压迫性狭窄。在术后短时间内，即术后2天至6个月内，其余7例表现为明显的气管狭窄，黏膜无刺激迹象。这些狭窄仅自1965年起有记录，即在我们诊所对每例喉切除术普遍采用吹入麻醉之后。这些观察结果使人们有理由认为麻醉操作应被视为气管狭窄发生的原因。在手术过程中的不利情况下，即使插管时间很短，除了其他众所周知的因素外，气管导管套囊的致压效应也可能对气管壁造成损害。
