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Mechanism of action and pharmacology studies with DTIC (NSC-45388).

作者信息

Loo T L, Housholder G E, Gerulath A H, Saunders P H, Farquhar D

出版信息

Cancer Treat Rep. 1976 Feb;60(2):149-52.

PMID:1260772
Abstract
摘要

相似文献

1
Mechanism of action and pharmacology studies with DTIC (NSC-45388).达卡巴嗪(NSC-45388)的作用机制及药理学研究
Cancer Treat Rep. 1976 Feb;60(2):149-52.
2
Studies on the mechanism of action of DTIC (NSC-45388).氮烯咪胺(NSC - 45388)作用机制的研究。
Cancer Treat Rep. 1976 Feb;60(2):141-8.
3
Effects of 5-(3,3-dimethyl-1-triazeno)imidazole-4-carboxamide and its metabolites on Novikoff hepatoma cells.5-(3,3-二甲基-1-三氮烯基)咪唑-4-甲酰胺及其代谢产物对诺维科夫肝癌细胞的影响。
Cancer Res. 1976 Aug;36(8):2827-31.
4
In vitro effect of sodium warfarin on DNA and RNA synthesis of mouse L1210 leukemic cells and Walker tumor cells.华法林钠对小鼠L1210白血病细胞和Walker肿瘤细胞DNA及RNA合成的体外作用
Oncology. 1973;28(3):232-7. doi: 10.1159/000224819.
5
The basis for the disparate sensitivity of L1210 leukemia and Walker 256 carcinoma to a new triazine folate antagonist.
Cancer Res. 1973 Nov;33(11):2972-6.
6
Pharmacology of a new triazine antifolate in mice, rats, dogs, and monkeys.一种新型三嗪类抗叶酸剂在小鼠、大鼠、狗和猴子体内的药理学研究
Cancer Res. 1975 Jan;35(1):17-22.
7
In vitro generation of a highly immunogenic subline of L1210 leukemia following exposure to 5-(3,3'-dimethyl-1-triazeno)imidazole-4-carboxamide.暴露于5-(3,3'-二甲基-1-三氮烯基)咪唑-4-甲酰胺后体外产生L1210白血病的高免疫原性子系。
Cancer Res. 1981 Jun;41(6):2476-82.
8
Effects of 5-fluorouracil on 5-fluorodeoxyuridine 5'-monophosphate and 2-deoxyuridine 5'-monophosphate pools, and DNA synthesis in solid mouse L1210 and rat Walker 256 tumors.
Cancer Res. 1978 Aug;38(8):2325-31.
9
Comparative pharmacology of pentamethylmelamine and hexamethylmelamine in mice.小鼠中五甲基三聚氰胺和六甲基三聚氰胺的比较药理学
Cancer Res. 1980 Aug;40(8 Pt 1):2762-7.
10
In vitro activation of dacarbazine (DTIC) for a human tumor cloning system.达卡巴嗪(DTIC)在人肿瘤克隆系统中的体外激活。
Int J Cell Cloning. 1983 Apr;1(1):24-32. doi: 10.1002/stem.5530010105.

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Adoptive cell therapy with CD4 T helper 1 cells and CD8 cytotoxic T cells enhances complete rejection of an established tumour, leading to generation of endogenous memory responses to non-targeted tumour epitopes.
采用CD4辅助性T细胞1和CD8细胞毒性T细胞进行过继性细胞治疗可增强对已形成肿瘤的完全排斥,从而产生针对非靶向肿瘤表位的内源性记忆反应。
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Oncolytic adenovirus expressing interleukin-18 improves antitumor activity of dacarbazine for malignant melanoma.表达白细胞介素-18的溶瘤腺病毒增强达卡巴嗪对恶性黑色素瘤的抗肿瘤活性。
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Systemic Therapies for Advanced Pancreatic Neuroendocrine Tumors.晚期胰腺神经内分泌肿瘤的全身治疗
Hematol Oncol Clin North Am. 2016 Feb;30(1):119-33. doi: 10.1016/j.hoc.2015.09.005. Epub 2015 Oct 23.
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Br J Cancer. 1993 Feb;67(2):362-8. doi: 10.1038/bjc.1993.66.
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A phase I clinical and pharmacological profile of dacarbazine with autologous bone marrow transplantation in patients with solid tumors.达卡巴嗪联合自体骨髓移植治疗实体瘤患者的I期临床及药理学研究
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9
Quantitation of drug sensitivity by human metastatic melanoma colony-forming units.通过人转移性黑色素瘤集落形成单位对药物敏感性进行定量分析。
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10
Pharmacokinetics of dacarbazine (DTIC) and its metabolite 5-aminoimidazole-4-carboxamide (AIC) following different dose schedules.达卡巴嗪(DTIC)及其代谢产物5-氨基咪唑-4-甲酰胺(AIC)在不同给药方案后的药代动力学。
Cancer Chemother Pharmacol. 1982;9(2):103-9. doi: 10.1007/BF00265388.