Meurer K A, Kloos G, Helber A, Kaufmann W
Klin Wochenschr. 1976 Mar 15;54(6):261-5. doi: 10.1007/BF01468921.
In patients with primary hypertension, intravenous administration of 150 mug Clonidin caused a decrease in arterial pressure, renin activity, RPF, GFR and electrolyte excretion. Activation of intracerebral adrenergic alpha-receptors and diminished sympathetic outflow seem to be responsible for the decrease of renin release.
在原发性高血压患者中,静脉注射150微克可乐定可导致动脉压、肾素活性、肾血浆流量、肾小球滤过率和电解质排泄减少。脑内肾上腺素能α受体的激活和交感神经输出减少似乎是肾素释放减少的原因。