Hertz C W
Postgrad Med J. 1976 Apr;52(606):209-17. doi: 10.1136/pgmj.52.606.209.
In pulmonary congestion dyspnoea is due not only to an increase in elastic but also in viscous work. Increased airway resistance can be reduced in most of these cases by inhalation of adrenergic or anti-cholinergic agents. Therefore, besides bronchial mucosal swelling induced by congestion in the bronchial vessels via bronchopulmonary anastomoses, bronchospasm can take place and lead to cardiac asthma. The possible genesis of this bronchoconstriction is discussed.
在肺充血时,呼吸困难不仅是由于弹性功增加,而且也是由于粘性功增加所致。在大多数这类病例中,吸入肾上腺素能或抗胆碱能药物可降低气道阻力增加。因此,除了通过支气管肺吻合支导致支气管血管充血引起的支气管粘膜肿胀外,还可能发生支气管痉挛并导致心源性哮喘。本文讨论了这种支气管收缩的可能发生机制。