Thiede W H, Manley J
Aviat Space Environ Med. 1976 May;47(5):553-5.
A case of cerebral air embolism responding immediately to compression to 6 atm is described. The patient, however, developed apparent cerebral edema while being decompressed. Subsequent recompression on oxygen was carried out twice daily at 60 ft (18 m, 2.8 ATA) for 60 min until the patient's symptoms had nearly cleared. While a number of other medications known to affect intracranial pressure were used, objective clinical improvement was noted only after hyperbaric oxygen. A question is raised of whether there is an association between the development of cerebral edema and the administration of intravenous 5% glucose in water.
本文描述了一例脑空气栓塞患者，对其施加6个大气压的压力后症状立即得到缓解。然而，该患者在减压过程中出现了明显的脑水肿。随后，每天两次在60英尺（18米，2.8个绝对大气压）的深度进行60分钟的吸氧再加压治疗，直至患者症状基本消失。虽然使用了许多其他已知会影响颅内压的药物，但仅在进行高压氧治疗后才观察到客观的临床改善。有人提出疑问，脑水肿的发生与静脉输注5%葡萄糖溶液之间是否存在关联。
