Alabovskiĭ V V, Vinokurov A A
Fiziol Zh (1978). 1992 Jul-Aug;38(4):15-9.
Reperfusion of the heart 30 min. after ischemia causes slight recovery of contractility and content of macroergic compounds in the myocardium tissue. Recovery of perfusion by the hypercalcium medium (0.05 mol/l) improves metabolism of the myocardium 30 min after ischemia. However, further perfusion by solution with physiological content of Ca2+ is followed by the development of the myocardium contracture, essential decrease in extracellular concentration of ATP and phosphocreatine. An increase in the extracellular sodium concentration and addition of macroergic compounds (ATR, phosphocreatine) adenosine, when reperfusing the heart by hypocalcium solution, improve the postischemic state of the myocardium and protect it from injuries during the following recovery of physiological Ca2+ content in the extracellular medium.
缺血30分钟后心脏再灌注可使心肌组织的收缩力和高能化合物含量稍有恢复。用高钙培养基（0.05 mol/l）恢复灌注可改善缺血30分钟后心肌的代谢。然而，随后用钙离子生理含量的溶液进行灌注会导致心肌挛缩的发生，细胞外ATP和磷酸肌酸浓度显著降低。当用低钙溶液对心脏进行再灌注时，增加细胞外钠浓度并添加高能化合物（三磷酸腺苷、磷酸肌酸）、腺苷，可改善心肌的缺血后状态，并在细胞外介质中生理钙离子含量随后恢复的过程中保护心肌免受损伤。
