牛磺酸氯胺抑制肺泡巨噬细胞中炎症介质的产生和诱导型一氧化氮合酶基因表达；两条途径的故事：第一部分，核因子κB信号传导 - Suppr

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超能文献

Taurine chloramine inhibits production of inflammatory mediators and iNOS gene expression in alveolar macrophages; a tale of two pathways: part I, NF-kappaB signaling.

作者信息

Quinn Michael R, Barua Madhabi, Liu Yong, Serban Valeria

机构信息

Center for Developmental Neuroscience, Staten Island, NY, USA.

出版信息

Adv Exp Med Biol. 2003;526:341-8. doi: 10.1007/978-1-4615-0077-3_42.

DOI:10.1007/978-1-4615-0077-3_42

PMID:12908618

Abstract

摘要

相似文献

Taurine chloramine inhibits production of inflammatory mediators and iNOS gene expression in alveolar macrophages; a tale of two pathways: part I, NF-kappaB signaling.牛磺酸氯胺抑制肺泡巨噬细胞中炎症介质的产生和诱导型一氧化氮合酶基因表达；两条途径的故事：第一部分，核因子κB信号传导

Adv Exp Med Biol. 2003;526:341-8. doi: 10.1007/978-1-4615-0077-3_42.

Taurine chloramine inhibits production of inflammatory mediators and iNOS gene expression in alveolar macrophages; a tale of two pathways: part II, IFN-gamma signaling through JAK/Stat.牛磺酸氯胺抑制肺泡巨噬细胞中炎症介质的产生和诱导型一氧化氮合酶基因表达；两条途径的故事：第二部分，通过JAK/Stat的γ-干扰素信号传导

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Taurine chloramine inhibits inducible nitric oxide synthase and TNF-alpha gene expression in activated alveolar macrophages: decreased NF-kappaB activation and IkappaB kinase activity.牛磺酸氯胺抑制活化肺泡巨噬细胞中诱导型一氧化氮合酶和肿瘤坏死因子-α基因的表达：降低核因子-κB激活及IκB激酶活性。

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Meconium induces expression of inducible NO synthase and activation of NF-kappaB in rat alveolar macrophages.胎粪可诱导大鼠肺泡巨噬细胞中诱导型一氧化氮合酶的表达及核因子κB的激活。

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Acetaminophen inhibits iNOS gene expression in RAW 264.7 macrophages: differential regulation of NF-kappaB by acetaminophen and salicylates.对乙酰氨基酚抑制RAW 264.7巨噬细胞中诱导型一氧化氮合酶（iNOS）基因表达：对乙酰氨基酚和水杨酸盐对核因子κB（NF-κB）的差异调节

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Hyaluronan fragments induce nitric-oxide synthase in murine macrophages through a nuclear factor kappaB-dependent mechanism.透明质酸片段通过核因子κB依赖性机制诱导小鼠巨噬细胞中的一氧化氮合酶。

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Increased nitric oxide synthase in the lung after ozone inhalation is associated with activation of NF-kappa B.吸入臭氧后肺中一氧化氮合酶增加与核因子κB的激活有关。

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Production of inflammatory mediators by activated C6 cells is attenuated by taurine chloramine inhibition of NF-kappaB activation.通过牛磺酸氯胺抑制NF-κB激活，可减弱活化的C6细胞产生炎性介质的能力。

Adv Exp Med Biol. 2003;526:365-72. doi: 10.1007/978-1-4615-0077-3_45.

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