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dy基因座的两个等位基因对小鼠垂体-睾丸轴的不同影响。

Differential effects of two alleles of the dy locus on the pituitary-testicular axis of mice.

作者信息

Amador A G, Mayerhofer A, Parkening T A, Collins T J, Bartke A

机构信息

Department of Obstetrics & Gynecology, SIU School of Medicine, Springfield 62794-9230.

出版信息

Rev Esp Fisiol. 1992 Sep;48(3):157-66.

PMID:1301630
Abstract

Testicular function was studied in vivo and in vitro in adult male dy/dy and dy2J/dy2J dystrophic mice. The results demonstrate that testicular function in dy/dy mice is more affected. The basal levels of pituitary hormones measured were normal in dystrophic mice, except for the presence of hyperprolactinemia in dy/dy mice. In dy/dy mice testicular weight was diminished and a deficient transduction of the gonadotropic signal is present in vivo, accompanied by reduced efficiency of 17-hydroxylase and 17-hydroxysteroid dehydrogenase. In dy2J/dy2J mice the signal transduction is normal and the reduction in enzyme efficiency is limited to 17-hydroxysteroid dehydrogenase. The in vitro HCG-induced increases in production of testosterone (T) and estradiol (E2) were reduced in dy/dy/mice, and the data indicate a reduction of enzyme activity rather than in efficiency. In dy21/dy21/mice, HCG-induced T synthesis was increased, HCG-induced E2 synthesis was normal, but basal media E2 levels were reduced, with the in vitro efficiency of aromatase being suppressed under both basal and HCG-stimulated conditions, when compared to their normal littermates.

摘要

在成年雄性dy/dy和dy2J/dy2J营养不良小鼠体内和体外研究了睾丸功能。结果表明,dy/dy小鼠的睾丸功能受影响更大。除dy/dy小鼠存在高催乳素血症外,营养不良小鼠所测垂体激素的基础水平正常。在dy/dy小鼠中,睾丸重量减轻,体内促性腺激素信号转导存在缺陷,同时17-羟化酶和17-羟类固醇脱氢酶的效率降低。在dy2J/dy2J小鼠中,信号转导正常,酶效率降低仅限于17-羟类固醇脱氢酶。在dy/dy小鼠中,体外人绒毛膜促性腺激素(HCG)诱导的睾酮(T)和雌二醇(E2)生成增加减少,数据表明是酶活性降低而非效率降低。在dy21/dy21小鼠中,HCG诱导的T合成增加,HCG诱导的E2合成正常,但基础培养基E2水平降低,与正常同窝小鼠相比,在基础和HCG刺激条件下,体外芳香化酶效率均受到抑制。

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