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The changing morphologic picture of endocarditis since the advent of chemotherapy and antibiotic agents.

作者信息

ANGRIST A, MARQUISS J

出版信息

Am J Pathol. 1954 Jan-Feb;30(1):39-63.

PMID:13124459
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1942455/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/e58f30719af4/amjpathol00701-0073-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/8dfca70e083e/amjpathol00701-0067-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/6bac9d751b22/amjpathol00701-0067-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/2fe89a15b1cf/amjpathol00701-0069-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/98a553b92498/amjpathol00701-0069-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/89fc4a2d5ed7/amjpathol00701-0070-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/447d4aa09d0f/amjpathol00701-0071-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/e58f30719af4/amjpathol00701-0073-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/8dfca70e083e/amjpathol00701-0067-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/6bac9d751b22/amjpathol00701-0067-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/2fe89a15b1cf/amjpathol00701-0069-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/98a553b92498/amjpathol00701-0069-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/89fc4a2d5ed7/amjpathol00701-0070-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/447d4aa09d0f/amjpathol00701-0071-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4e1d/1942455/e58f30719af4/amjpathol00701-0073-a.jpg

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The changing morphologic picture of endocarditis since the advent of chemotherapy and antibiotic agents.自化疗和抗生素药物问世以来心内膜炎形态学表现的变化。
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Studies in experimental endocarditis. I. Production of valvular lesions by mechanisms not involving infection or sensitivity factors.实验性心内膜炎研究。I. 不涉及感染或敏感因素机制的瓣膜病变产生。

本文引用的文献

1
The Fate of Endocardial Vegetations Following Penicillin Treatment of Bacterial Endocarditis.青霉素治疗细菌性心内膜炎后心内膜赘生物的转归
Am J Pathol. 1947 Nov;23(6):1023-35.
2
The Rôle Played by Rheumatic Fever in the Implantation of Bacterial Endocarditis.风湿热在细菌性心内膜炎发病过程中所起的作用。
Am J Pathol. 1937 Sep;13(5):769-798.9.
3
[Immunity characteristic of postwar endocarditis lenta].[战后亚急性细菌性心内膜炎的免疫特征]
Am J Pathol. 1960 Feb;36(2):181-99.
4
The nature of arteriolar and capillary occlusion in patients with carcinoma.癌症患者小动脉和毛细血管阻塞的性质。
Am J Pathol. 1956 Nov-Dec;32(6):1185-203.
5
Bacterial endocarditis in cirrhosis.肝硬化患者的细菌性心内膜炎
Dig Dis Sci. 1981 Oct;26(10):935-7. doi: 10.1007/BF01309500.
6
Staphylococcus aureus bacteremia: A critical review of its treatment and association with infective endocarditis.金黄色葡萄球菌菌血症：对其治疗及与感染性心内膜炎关联的批判性综述
Infection. 1973;1(4):214-7. doi: 10.1007/BF01639652.
Z Gesamte Inn Med. 1952 Apr 15;7(8):337-40.
4
[Endocarditis lenta as the sequel of a primary metabolic disorder (dysproteinemia)].[迟发性心内膜炎作为原发性代谢紊乱（异常蛋白血症）的后遗症]
Med Klin. 1952 Jan 18;47(3):69-71.
5
[Endocarditis lenta as reactive reticulosis with special reference to liver changes].[慢性心内膜炎作为反应性网状细胞增多症，特别提及肝脏变化]
Dtsch Med Wochenschr. 1952 Feb 29;77(9):259-64. doi: 10.1055/s-0028-1115931.
6
Bacterial endocarditis.细菌性心内膜炎
Am Heart J. 1951 Sep;42(3):472-82. doi: 10.1016/0002-8703(51)90144-5.
7
Bacterial endocarditis: clinicopathologic studies of untreated, treated and cured patients.细菌性心内膜炎：未经治疗、接受治疗及治愈患者的临床病理研究
Ann Intern Med. 1951 Jul;35(1):45-58. doi: 10.7326/0003-4819-35-1-45.
8
A new method for the production of experimental bacterial endocarditis.一种制备实验性细菌性心内膜炎的新方法。
Proc Soc Exp Biol Med. 1950 Nov;75(2):573-7. doi: 10.3181/00379727-75-18270.
9
The occurrence of endocarditis with valvular deformities in dogs with arteriovenous fistulas.患有动静脉瘘的犬类出现伴有瓣膜畸形的心内膜炎。
Ann Surg. 1950 Oct;132(4):577-90. doi: 10.1097/00000658-195010000-00001.
10
A study of the endocardial lesions of subacute bacterial endocarditis; with particular reference to healing or healed lesions; with clinical notes.亚急性细菌性心内膜炎的心内膜病变研究；特别涉及愈合或已愈合的病变；附临床记录。
Am J Med. 1952 Nov;13(5):544-9. doi: 10.1016/0002-9343(52)90018-1.