Suzuki K, Takatsu T, Inoue H, Teramoto T, Ishida Y, Ohmori K
Department of Orthopaedic and Spinal Surgery, Nagoya Daini Red Cross Hospital, Japan.
Spine (Phila Pa 1976). 1992 Nov;17(11):1337-42. doi: 10.1097/00007632-199211000-00013.
To investigate pathogenesis of redundant nerve roots of the cauda equina, which were concomitant with severe lumbar spinal canal stenosis, six cadavers were examined anatomically and histopathologically, and quantitative analysis of nerve fibers was performed. In this anatomic study, it was observed that all the redundant nerve roots passed through the constriction of the spinal canal. No significant pathologic change was detected in the spinal ganglia and in the spinal cords except for the posterior column, in which dorsal redundant roots were entering. Redundant nerve roots of unequal length also were observed in the anatomic study. The spatial distribution of redundant nerve roots and the extent of degeneration of nerve fibers in them were established by these histopathologic and quantitative studies. These facts indicated a close causal relationship between redundant nerve roots and constriction of the spinal canal, and that the pathogenesis of redundant nerve roots was a squeezing force acting on the nerve roots at the area of spinal canal constriction.
为研究伴有严重腰椎管狭窄的马尾神经根冗余的发病机制，对6具尸体进行了解剖学和组织病理学检查，并对神经纤维进行了定量分析。在这项解剖学研究中，观察到所有冗余神经根均通过椎管狭窄处。除后柱（即背侧冗余神经根进入处）外，脊髓神经节和脊髓未检测到明显的病理变化。解剖学研究中还观察到长度不等的冗余神经根。通过这些组织病理学和定量研究确定了冗余神经根的空间分布及其神经纤维的退变程度。这些事实表明冗余神经根与椎管狭窄之间存在密切的因果关系，且冗余神经根的发病机制是椎管狭窄区域作用于神经根的挤压力量。
