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相似文献

1
Stuart clotting defect. I. Segregation of an hereditary hemorrhagic state from the heterogeneous group heretofore called stable factor (SPCA, proconvertin, factor VII) deficiency.斯图尔特凝血缺陷。I. 从迄今为止称为稳定因子（血清凝血酶原转变加速素、前转变素、因子VII）缺乏的异质性组中分离出一种遗传性出血状态。
J Clin Invest. 1957 Mar;36(3):485-96. doi: 10.1172/JCI103446.
2
Some biochemical and electrophoretic studies on purified prothrombin, factor VII (proconvertin) and factor X (Stuart).关于纯化的凝血酶原、因子VII（前转变素）和因子X（斯图尔特因子）的一些生化及电泳研究。
Blood. 1960 May;15:778-90.
3
[Reflections on the use of PPSB fraction (Prothrombin-proconvertin-Stuart factor-antihemophilic factor B)].[关于凝血酶原-前转变素-斯图尔特因子-抗血友病因子B（PPSB）组分使用的思考]
Transfusion (Paris). 1966;9(3):245-54.
4
The role of proconvertin and Stuart factor in the inactivation of tissue thromboplastin by serum.
Experientia. 1960 Oct 15;16:455-6. doi: 10.1007/BF02171149.
5
[Erythroplastin: coagulant factor of the erythrocytes. Relation to proaccelerin, proconvertin and the Stuart factor].[促红细胞凝血素：红细胞的凝血因子。与前加速素、前转变素及斯图尔特因子的关系]
Pathol Biol. 1961 Sep;9:1735-8.
6
[Preparation of a fraction rich in prothrombin, proconvertin, Stuart factor and antihemophilic factor B (P.P.B. fraction)].富含凝血酶原、凝血激酶原、斯图尔特因子和抗血友病因子B的组分（P.P.B.组分）的制备
Pathol Biol. 1959 Dec;7:2477-86.
7
Blood coagulation as a continuous process. II. The turnover rate of proconvertin.血液凝固作为一个连续过程。II. 凝血酶原转变加速因子的周转率。
Acta Med Scand. 1960 Dec 20;168:477-82.
8
The fate of factor VII and Stuart factor during the clotting of normal blood.正常血液凝固过程中因子VII和斯图尔特因子的命运。
Thromb Diath Haemorrh. 1959 Jun 15;3:367-74.
9
[Hemorrhagic diathesis caused by factor VII (proconvertin) deficiency].
Probl Oncol. 1961 Mar;6:22-30.
10
Congenital bleeding disorders of the vitamin K-dependent clotting factors.维生素K依赖凝血因子的先天性出血性疾病。
Vitam Horm. 2008;78:281-374. doi: 10.1016/S0083-6729(07)00014-3.

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1
Congenital Factor X-Riyadh (Stuart-Prower) Deficiency With Isolated Prothrombin Time Prolongation: A Case Report.先天性因子X-利雅得（斯图尔特-普罗沃）缺乏伴孤立性凝血酶原时间延长：一例报告
Cureus. 2023 Apr 12;15(4):e37488. doi: 10.7759/cureus.37488. eCollection 2023 Apr.
2
[Congenital factor X deficiency: a retrospective analysis of 11 cases].先天性因子X缺乏症：11例回顾性分析
Zhonghua Xue Ye Xue Za Zhi. 2022 Jan 14;43(1):26-30. doi: 10.3760/cma.j.issn.0253-2727.2022.01.006.
3
Blood coagulation factor X: molecular biology, inherited disease, and engineered therapeutics.凝血因子 X：分子生物学、遗传性疾病与工程治疗学。
J Thromb Thrombolysis. 2021 Aug;52(2):383-390. doi: 10.1007/s11239-021-02456-w. Epub 2021 Apr 22.
4
Structure and function of factor X: properties, activation, and activity in prothrombinase. A retrospective in a historical context.因子 X 的结构和功能：在凝血酶原酶中的特性、激活和活性。历史背景下的回顾。
J Thromb Thrombolysis. 2021 Aug;52(2):371-378. doi: 10.1007/s11239-021-02421-7. Epub 2021 Mar 16.
5
The Effect of Polyphenols on Hypercholesterolemia through Inhibiting the Transport and Expression of Niemann-Pick C1-Like 1.多酚通过抑制 Niemann-Pick C1-like 1 的转运和表达对高胆固醇血症的影响。
Int J Mol Sci. 2019 Oct 6;20(19):4939. doi: 10.3390/ijms20194939.
6
Transporters for the Intestinal Absorption of Cholesterol, Vitamin E, and Vitamin K.胆固醇、维生素E和维生素K肠道吸收的转运蛋白
J Atheroscler Thromb. 2017 Apr 3;24(4):347-359. doi: 10.5551/jat.RV16007. Epub 2017 Jan 17.
7
Factor X Friuli Coagulation Disorder: Almost 50 Years Later.弗留利X因子凝血障碍：近50年后
Clin Appl Thromb Hemost. 2018 Jan;24(1):33-40. doi: 10.1177/1076029616686423. Epub 2016 Dec 29.
8
Surgical treatment for a paraplegic patient induced by congenital factor X deficiency.先天性因子X缺乏所致截瘫患者的外科治疗
Int J Clin Exp Med. 2015 Aug 15;8(8):13403-7. eCollection 2015.
9
Successful treatment of a noninhibitory antibody-mediated acquired factor X deficiency in a patient with marginal-zone lymphoma.成功治疗一名边缘区淋巴瘤患者的非抑制性抗体介导的获得性因子X缺乏症。
Clin Case Rep. 2015 Jul;3(7):587-93. doi: 10.1002/ccr3.294. Epub 2015 May 25.
10
Inherited Factor X (Stuart-Prower Factor) deficiency and its management.遗传性因子X（斯图尔特-普劳尔因子）缺乏症及其管理。
Med J Armed Forces India. 2015 Jul;71(Suppl 1):S184-6. doi: 10.1016/j.mjafi.2014.01.007. Epub 2014 Apr 3.

本文引用的文献

1
Hypoprothrombinemia; studies of a case of the idiopathic type and the effect of serum administration.低凝血酶原血症；特发性低凝血酶原血症一例的研究及血清输注的效果
Blood. 1949 Dec;4(12):1298-1309.
2
Congenital SPCA deficiency: a hitherto unrecognized coagulation defect with hemorrhage rectified by serum and serum fractions.先天性血清凝血活酶原复合物缺乏症：一种迄今未被认识的凝血缺陷，可通过血清和血清成分纠正出血。
J Clin Invest. 1951 Jun;30(6):596-608. doi: 10.1172/JCI102477.
3
Two cases of congenital hypoconvertinemia.两例先天性低转化素血症。
Sang. 1955;26(3):315-21.
4
A simple, specific one-stage prothrombin assay using Russell's viper venom in cephalin suspension.一种在脑磷脂悬浮液中使用罗素蝰蛇毒的简单、特异的一步法凝血酶原测定法。
J Lab Clin Med. 1955 Jul;46(1):89-97.
5
Plasma thromboplastin component (PTC) deficiency produced by prolonged administration or prothrombopenic anticoagulants.长期使用促凝血酶原减少性抗凝剂导致血浆凝血活酶成分（PTC）缺乏。
Proc Soc Exp Biol Med. 1955 May;89(1):81-3. doi: 10.3181/00379727-89-21721.
6
[Congenital hypoproconvertinemia].[先天性低凝血酶原转变素血症]
Acta Haematol. 1955 Apr;13(4):242-9. doi: 10.1159/000204944.
7
A familial hemorrhagic trait associated with a deficiency of a clot-promoting fraction of plasma.一种与血浆中促凝成分缺乏相关的家族性出血性状。
J Clin Invest. 1955 Apr;34(4):602-13. doi: 10.1172/JCI103109.
8
Stuart clotting defect. II. Genetic aspects of a new hemorrhagic state.斯图尔特凝血缺陷。二、一种新的出血状态的遗传学方面。
J Clin Invest. 1957 Mar;36(3):497-503. doi: 10.1172/JCI103447.
9
[Congenital factor VII (SPCA) deficiency as the cause of hemophilic-type hemorrhagic diathesis].先天性因子VII（SPCA）缺乏作为血友病样出血素质的病因
Acta Haematol. 1956 Sep;16(3):181-98. doi: 10.1159/000205126.
10
A new coagulation defect.一种新的凝血缺陷。
Br J Haematol. 1956 Jul;2(3):308-16. doi: 10.1111/j.1365-2141.1956.tb06703.x.

Stuart clotting defect. I. Segregation of an hereditary hemorrhagic state from the heterogeneous group heretofore called stable factor (SPCA, proconvertin, factor VII) deficiency.

作者信息

HOUGIE C, BARROW E M, GRAHAM J B

出版信息

J Clin Invest. 1957 Mar;36(3):485-96. doi: 10.1172/JCI103446.

DOI:10.1172/JCI103446
PMID:13406063
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1072666/
Abstract
摘要