Overproduction of uric acid as the cause of hyperuricemia in primary gout.
作者信息
WYNGAARDEN J B
出版信息
J Clin Invest. 1957 Oct;36(10):1508-15. doi: 10.1172/JCI103547.
Abstract
摘要
相似文献
1
Overproduction of uric acid as the cause of hyperuricemia in primary gout.原发性痛风中尿酸生成过多作为高尿酸血症的病因。
J Clin Invest. 1957 Oct;36(10):1508-15. doi: 10.1172/JCI103547.
2
[Considerations on the pathogenesis of hyperuricemia in the gout patient].[关于痛风患者高尿酸血症发病机制的思考]
Viata Med. 1961 May 15;8:551-4.
3
Heredity in gout and hyperuricemia.痛风与高尿酸血症中的遗传因素。
Acta Med Scand. 1955 Nov 10;152(4):247-57. doi: 10.1111/j.0954-6820.1955.tb03485.x.
4
[Uptake of 2-C14 urate by erythrocytes in hyperuricemia and in gout (author's transl)].高尿酸血症和痛风患者红细胞对2-C14尿酸的摄取（作者译）
Klin Wochenschr. 1978 Mar 15;53(16):261-4.
5
[Hyperlipoproteinemia in primary gout and asymptomatic hyperuricemia].
Wien Med Wochenschr. 1988 May 31;138(10):221-5.
6
A new standard of care? Studies on febuxostat in the management of hyperuricemia with and without gout.一种新的治疗标准？非布司他治疗伴或不伴痛风的高尿酸血症的研究。
J Clin Rheumatol. 2011 Jun;17(4 Suppl 2):S11-2. doi: 10.1097/RHU.0b013e31821d479d.
7
8
Placebo-controlled double-blind dose-response study of the non-purine-selective xanthine oxidase inhibitor febuxostat (TMX-67) in patients with hyperuricemia (including gout patients) in japan: late phase 2 clinical study.安慰剂对照、双盲、剂量反应研究日本高尿酸血症（包括痛风患者）患者中黄嘌呤氧化酶抑制剂非嘌呤选择性抑制剂 febuxostat（TMX-67）：第 2 期后期临床研究。
J Clin Rheumatol. 2011 Jun;17(4 Suppl 2):S35-43. doi: 10.1097/RHU.0b013e31821d351d.
9
[The role of the kidney in hyperuricemia from the study of 390 clearances of uric acid].[通过390例尿酸清除率研究看肾脏在高尿酸血症中的作用]
Rev Rhum Mal Osteoartic. 1955 Apr;22(4):320-9.
10
Urate testing in gout: why, when and how.痛风中的尿酸检测：为何、何时及如何进行检测
N Z Med J. 2015 Aug 21;128(1420):65-8.
引用本文的文献
1
Variation of urate transport in the nephrons in subtypes of hyperuricemia.高尿酸血症各亚型中肾单位尿酸转运的变化
Nephron Extra. 2013 Aug 20;3(1):73-85. doi: 10.1159/000354029. eCollection 2013.
2
Lack of Correlation Between Gout and the Incorporation of Isotopic Formate into Uric Acid.痛风与同位素甲酸掺入尿酸之间缺乏相关性。
Yale J Biol Med. 1961 Aug;34(1):31-9.
3
The effects of azathioprine (imuran) on purine synthesis in clinical disorders of purine metabolism.巯嘌呤（依木兰）对嘌呤代谢临床紊乱中嘌呤合成的影响。
J Clin Invest. 1967 Sep;46(9):1518-29. doi: 10.1172/JCI105643.
4
Gout and hyperuricaemia in rural and urban populations.农村和城市人口中的痛风与高尿酸血症
Ann Rheum Dis. 1962 Jun;21(2):154-63. doi: 10.1136/ard.21.2.154.
5
Accelerated turnover of phosphoribosylpyrophosphate, a purine nucleotide precursor, in certain gouty subjects.在某些痛风患者中，嘌呤核苷酸前体磷酸核糖焦磷酸的周转加速。
J Clin Invest. 1962 Sep;41(9):1805-15. doi: 10.1172/JCI104638.
6
A quantitative study of recycling of isotope from glycine-1-C14, alpha-N15 into various subunits of the uric acid molecule in a normal subject.对一名正常受试者体内甘氨酸 -1-C14、α-N15的同位素再循环进入尿酸分子各个亚基的定量研究。
J Clin Invest. 1961 Nov;40(11):2076-82. doi: 10.1172/JCI104433.
7
The renal excretion of uric acid in patients with gout and in nongouty subjects.痛风患者和非痛风患者的尿酸肾脏排泄情况。
J Clin Invest. 1959 Nov;38(11):1890-8. doi: 10.1172/JCI103966.
8
Suppression of uric acid synthesis in the gouty human by the use of 6-diazo-5-oxo-L-norleucine.使用6-重氮-5-氧代-L-正亮氨酸对痛风患者尿酸合成的抑制作用。
J Clin Invest. 1960 Mar;39(3):447-54. doi: 10.1172/JCI104057.
9
Uric acid production in gout.痛风中的尿酸生成
J Clin Invest. 1961 Jul;40(7):1304-14. doi: 10.1172/JCI104360.
10
Uric acid metabolism in psoriasis.银屑病中的尿酸代谢
J Clin Invest. 1961 Aug;40(8 Pt 1-2):1486-94. doi: 10.1172/JCI104379.
本文引用的文献
1
Windowless, Flow Type, Proportional Counter for Counting C14.用于计数碳-14的无窗流动型正比计数器。
Science. 1950 Aug 18;112(2903):198-9. doi: 10.1126/science.112.2903.198.
2
Increased renal excretion of urate in young patients with gout.痛风年轻患者尿酸肾排泄增加。
Am J Med. 1950 Jul;9(1):31-4. doi: 10.1016/0002-9343(50)90005-2.
3
Effect of benemid (p-[di-n-propylsulfamyl]-benzoic acid) on urate clearance and other discrete renal functions in gouty subjects.丙磺舒（对-[二正丙基氨磺酰基]-苯甲酸）对痛风患者尿酸清除率及其他离散肾功能的影响。
J Clin Invest. 1952 Jul;31(7):692-701. doi: 10.1172/JCI102651.
4
Incorporation of glycine nitrogen into uric acid in normal and gouty man.正常人和痛风患者体内甘氨酸氮掺入尿酸的情况。
Metabolism. 1952 Jan;1(1):3-12.
5
The biosynthesis of nucleic acid components studied with C14. I. Purines and pyrimidines in the rat.用C14研究核酸成分的生物合成。I. 大鼠体内的嘌呤和嘧啶。
J Biol Chem. 1950 Oct;186(2):447-60.
6
The fate of uric acid in the normal and gouty human being.正常人和痛风患者体内尿酸的代谢情况。
J Chronic Dis. 1955 Jul;2(1):42-9. doi: 10.1016/0021-9681(55)90106-5.
7
8
Physiological disposition and metabolic fate of infused pentoses in man.人体内输注戊糖的生理处置和代谢命运
J Clin Invest. 1957 Oct;36(10):1395-407. doi: 10.1172/JCI103539.
9
Intermediary purine metabolism and the metabolic defects of gout.嘌呤中间代谢与痛风的代谢缺陷
Metabolism. 1957 May;6(3):244-68.
10
Uricolysis in normal and gouty individuals.正常人和痛风患者体内的尿酸分解
Ann Rheum Dis. 1955 Dec;14(4):409-11. doi: 10.1136/ard.14.4.409.
Zotero 插件