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Modulation of the expression of intercellular adhesion molecule-1 (ICAM-1) in human keratinocytes by ultraviolet (UV) radiation.

作者信息

Krutmann J, Trefzer U

机构信息

Department of Dermatology, University of Freiburg, Federal Republic of Germany.

出版信息

Springer Semin Immunopathol. 1992;13(3-4):333-44. doi: 10.1007/BF00200532.

DOI:10.1007/BF00200532
PMID:1357761
Abstract
摘要

相似文献

1
Modulation of the expression of intercellular adhesion molecule-1 (ICAM-1) in human keratinocytes by ultraviolet (UV) radiation.紫外线（UV）辐射对人角质形成细胞中细胞间黏附分子-1（ICAM-1）表达的调节作用。
Springer Semin Immunopathol. 1992;13(3-4):333-44. doi: 10.1007/BF00200532.
2
Contrasting effects of an ultraviolet B and an ultraviolet A tanning lamp on interleukin-6, tumour necrosis factor-alpha and intercellular adhesion molecule-1 expression.紫外线B和紫外线A晒黑灯对白介素-6、肿瘤坏死因子-α和细胞间黏附分子-1表达的对比作用
Br J Dermatol. 2001 Jul;145(1):54-62. doi: 10.1046/j.1365-2133.2001.04281.x.
3
Evidence that DNA damage is a mediate in ultraviolet B radiation-induced inhibition of human gene expression: ultraviolet B radiation effects on intercellular adhesion molecule-1 (ICAM-1) expression.DNA损伤是紫外线B辐射诱导人类基因表达抑制的介导因素的证据：紫外线B辐射对细胞间黏附分子-1（ICAM-1）表达的影响。
J Invest Dermatol. 1994 Apr;102(4):428-32. doi: 10.1111/1523-1747.ep12372947.
4
Tumor necrosis factor beta and ultraviolet radiation are potent regulators of human keratinocyte ICAM-1 expression.肿瘤坏死因子β和紫外线是人类角质形成细胞细胞间黏附分子-1表达的有效调节因子。
J Invest Dermatol. 1990 Aug;95(2):127-31. doi: 10.1111/1523-1747.ep12477839.
5
Ultraviolet radiation can either suppress or induce expression of intercellular adhesion molecule 1 (ICAM-1) on the surface of cultured human keratinocytes.
J Invest Dermatol. 1990 Aug;95(2):132-8. doi: 10.1111/1523-1747.ep12477877.
6
Ultraviolet radiation effects on human keratinocyte ICAM-1 expression: UV-induced inhibition of cytokine-induced ICAM-1 mRNA expression is transient, differentially restored for IFN gamma versus TNF alpha, and followed by ICAM-1 induction via a TNF alpha-like pathway.紫外线辐射对人角质形成细胞细胞间黏附分子-1表达的影响：紫外线诱导的细胞因子诱导的细胞间黏附分子-1 mRNA表达抑制是短暂的，对干扰素γ和肿瘤坏死因子α的恢复存在差异，随后通过类似肿瘤坏死因子α的途径诱导细胞间黏附分子-1。
J Invest Dermatol. 1992 Jun;98(6):923-8. doi: 10.1111/1523-1747.ep12460737.
7
Expression of intercellular adhesion molecule-1 (ICAM-1) and OKM5 in UVA- and UVB-induced lesions in patients with lupus erythematosus and polymorphous light eruption.细胞间黏附分子-1（ICAM-1）和OKM5在红斑狼疮和多形性日光疹患者UVA及UVB诱发皮损中的表达
Arch Dermatol Res. 1993;285(6):328-33. doi: 10.1007/BF00371832.
8
Involvement of cytokines, DNA damage, and reactive oxygen intermediates in ultraviolet radiation-induced modulation of intercellular adhesion molecule-1 expression.细胞因子、DNA损伤和活性氧中间体在紫外线辐射诱导的细胞间黏附分子-1表达调节中的作用。
J Invest Dermatol. 1995 Jul;105(1 Suppl):67S-70S. doi: 10.1111/1523-1747.ep12316095.
9
Differential effect of griseofulvin on interferon-gamma-induced HLA-DR and intercellular adhesion molecule-1 expression of human keratinocytes.灰黄霉素对干扰素-γ诱导的人角质形成细胞HLA-DR及细胞间黏附分子-1表达的差异影响。
Br J Dermatol. 1992 Sep;127(3):258-61. doi: 10.1111/j.1365-2133.1992.tb00124.x.
10
RNA released from necrotic keratinocytes upregulates intercellular adhesion molecule-1 expression in melanocytes.坏死角质形成细胞释放的 RNA 上调黑素细胞中细胞间黏附分子-1 的表达。
Arch Dermatol Res. 2011 Dec;303(10):771-6. doi: 10.1007/s00403-011-1170-8. Epub 2011 Aug 30.

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J Am Acad Dermatol. 2012 Oct;67(4):760-8. doi: 10.1016/j.jaad.2011.12.021. Epub 2012 Jan 30.
2
Understanding loss of donor white blood cell immunogenicity after pathogen reduction: mechanisms of action in ultraviolet illumination and riboflavin treatment.了解病原体减少处理后供体白细胞免疫原性丧失：紫外线照射和核黄素处理的作用机制。
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本文引用的文献

1
Ultraviolet radiation induces changes in membrane metabolism of human keratinocytes in culture.紫外线辐射会诱导培养的人角质形成细胞的膜代谢发生变化。
J Invest Dermatol. 1984 Nov;83(5):323-6. doi: 10.1111/1523-1747.ep12264114.
2
Xeroderma pigmentosum: a human disease in which an initial stage of DNA repair is defective.着色性干皮病：一种人类疾病，其DNA修复的初始阶段存在缺陷。
Proc Natl Acad Sci U S A. 1969 Jun;63(2):428-35. doi: 10.1073/pnas.63.2.428.
3
Chromosome mapping of cell membrane antigens expressed on activated B cells.
Eur J Immunol. 1985 Jan;15(1):103-6. doi: 10.1002/eji.1830150121.
4
Enhancement of the elicitation phase of the murine contact hypersensitivity response by prior exposure to local ultraviolet radiation.
J Invest Dermatol. 1986 Jan;86(1):13-7. doi: 10.1111/1523-1747.ep12283708.
5
A human intercellular adhesion molecule (ICAM-1) distinct from LFA-1.一种不同于淋巴细胞功能相关抗原-1的人类细胞间黏附分子（ICAM-1）。
J Immunol. 1986 Aug 15;137(4):1270-4.
6
Primary structure of ICAM-1 demonstrates interaction between members of the immunoglobulin and integrin supergene families.细胞间黏附分子-1的一级结构显示了免疫球蛋白超基因家族成员与整合素超基因家族成员之间的相互作用。
Cell. 1988 Mar 25;52(6):925-33. doi: 10.1016/0092-8674(88)90434-5.
7
ICAM, an adhesion ligand of LFA-1, is homologous to the neural cell adhesion molecule NCAM.细胞间黏附分子（ICAM）是淋巴细胞功能相关抗原-1（LFA-1）的一种黏附配体，与神经细胞黏附分子（NCAM）同源。
Nature. 1988 Feb 18;331(6157):624-7. doi: 10.1038/331624a0.
8
Role of interferon-gamma in cutaneous trafficking of lymphocytes with emphasis on molecular and cellular adhesion events.γ干扰素在淋巴细胞皮肤归巢中的作用，重点关注分子和细胞黏附事件。
Arch Dermatol. 1988 Dec;124(12):1835-43.
9
Adhesion of T lymphoblasts to epidermal keratinocytes is regulated by interferon gamma and is mediated by intercellular adhesion molecule 1 (ICAM-1).T淋巴母细胞与表皮角质形成细胞的黏附受γ干扰素调节，并由细胞间黏附分子1（ICAM-1）介导。
J Exp Med. 1988 Apr 1;167(4):1323-40. doi: 10.1084/jem.167.4.1323.
10
Induction by IL 1 and interferon-gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM-1).白细胞介素-1和γ干扰素诱导：一种天然黏附分子（细胞间黏附分子-1）的组织分布、生物化学及功能
J Immunol. 1986 Jul 1;137(1):245-54.