通过盐敏感突变体研究金黄色葡萄球菌的耐盐机制。
The salt-resistance mechanism of Staphylococcus aureus examined by salt-sensitive mutants.
作者信息
Kanemasa Y, Takatsu T, Sasai K, Kojima I, Hayashi H
出版信息
Acta Med Okayama. 1976 Aug;30(4):271-6.
PMID:137654
Abstract
Two NaCl-sensitive mutants of S. aureus 209P (S-16 and S-23) were isolated after treatment of the parental strain with N-methyl-N'-nitro-N-nitrosoguanidine. Examination of the lipid components revealed that S-16 contained no cardiolipin and that S-23 contained only a small amount of cardiolipin. An increase of cardiolipin was indispensable for S. aureus proliferation in high NaCl medium.
摘要
在用N-甲基-N'-硝基-N-亚硝基胍处理亲本菌株后，分离出了金黄色葡萄球菌209P的两个对氯化钠敏感的突变体（S-16和S-23）。对脂质成分的检测表明，S-16不含心磷脂，而S-23仅含有少量心磷脂。心磷脂的增加对于金黄色葡萄球菌在高氯化钠培养基中的增殖是必不可少的。
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