Bardy M, Tsacopoulos M, Barras J P
Arch Ophtalmol (Paris). 1976 Aug-Sep;36(8-9):605-14.
Experimental variations of the anterior chamber pressure (P.I.C.) are transmitted at about 300 ms to the middle of the vitreous. The response time to a sudden experimental increase in P.I.C. to the centre of the vitreous is variable and depends essentially on the control intra-ocular pressure: the more it is raised the shorter the response time. The pressure in the centre of the vitreous (P.I.V.) has a pulsatile character probably due to transmission of arterial pulsation. The higher the intra-ocular pressure the greater the amplitude of these pulsations. Recording of P.I.V. close to the surface of the retina show abrupt falls despite the fact that the P.I.C. and systemic arterial pressure remain stable. These results are discussed in relationship to autoregulation of the retinal circulation and the physiopathology of glaucoma.
前房压力（P.I.C.）的实验性变化在约300毫秒时传递至玻璃体中部。对P.I.C.突然实验性升高至玻璃体中心的反应时间是可变的，并且主要取决于眼内压的控制值：眼内压升得越高，反应时间越短。玻璃体中心的压力（P.I.V.）具有搏动性特征，这可能是由于动脉搏动的传递所致。眼内压越高，这些搏动的幅度就越大。尽管P.I.C.和全身动脉压保持稳定，但在靠近视网膜表面记录P.I.V.时仍显示出突然下降。结合视网膜循环的自动调节和青光眼的病理生理学对这些结果进行了讨论。
