Barabanova V V
Fiziol Zh SSSR Im I M Sechenova. 1977 Sep;63(9):1312-8.
The effect of the parathyroid hormone (PTH) on AP and the ventricle myocardium contractions in frogs was studied under conditions of the calcium permeability blockade and in calciumfree solution: the AP duration shortened, while AP amplitude increased like in control solution. The hormone's ion mechanisms are supposed to be based on increased transfer of calcium along the "slow" channels of membrane and of sodium--along the "fast" channels, as well as on activation of the Na-K-dependent ATPhase.
在钙通透性被阻断的条件下以及在无钙溶液中,研究了甲状旁腺激素(PTH)对青蛙动作电位(AP)和心室心肌收缩的影响:动作电位持续时间缩短,而动作电位幅度像在对照溶液中一样增加。该激素的离子机制被认为是基于钙沿膜的“慢”通道转运增加以及钠沿“快”通道转运增加,以及基于钠钾依赖性三磷酸腺苷酶(Na-K-ATPase)的激活。