Formation of malonaldehyde in vitamin E deficiency and its relation to the inhibition of gulonolactone oxidase. - Suppr | 超能文献

Formation of malonaldehyde in vitamin E deficiency and its relation to the inhibition of gulonolactone oxidase.

作者信息

KITABCHI A E, MCCAY P B, CARPENTER M P, TRUCCO R E, CAPUTTO R

出版信息

J Biol Chem. 1960 Jun;235:1591-8.

Abstract

摘要

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Formation of malonaldehyde in vitamin E deficiency and its relation to the inhibition of gulonolactone oxidase.

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Lipid peroxidation in rat tissue homogenates: Interaction of iron and ascorbic acid as the normal catalytic mechanism.大鼠组织匀浆中的脂质过氧化：铁与抗坏血酸的相互作用作为正常催化机制。

Lipids. 1966 Mar;1(2):146-51. doi: 10.1007/BF02533008.

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Biochemical changes in the tissues of animals injected with iron. 3. Lipid peroxidation.注射铁剂的动物组织中的生化变化。3. 脂质过氧化作用。

Biochem J. 1962 May;83(2):291-8. doi: 10.1042/bj0830291.

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Some hormonal, metabolic, and nutritional factors influencing lipid peroxidation by rat adipose tissue in vitro.一些激素、代谢和营养因素对大鼠脂肪组织体外脂质过氧化的影响。

J Clin Invest. 1962 Jun;41(6):1288-96. doi: 10.1172/JCI104591.

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Vitamin E, catalase, manganous or cobaltous ions and dithiothreitol protect against Tween 20-induced hemolysis of vitamin E-deficient chick and kid erythrocytes.

Experientia. 1984 Mar 15;40(3):258-9. doi: 10.1007/BF01947569.