Sherman D M
Patol Fiziol Eksp Ter. 2003 Jul-Sep(3):9-12.
Being directly, extremely dangerously affected, the organism of man (and laboratory animals) automatically starts showing an ancient hypobiotic protective reaction in the form of unique torpid syndrome. However, hypobiotic conditions are evolutionarily alien to the superior brain formations. A functional conflict arises between the new and old regulatory systems. Increasing pathology of regulation forms the whole typical stereotypical shock syndrome with its dangerous pathological components and phase course. However, shock maintains the organism's vitality and saves it from immediate death after the damaging effect of the initial collapse. The results depend on the adequacy of anti-shock therapy, the degree of organism's biological reserve preservation and severity of organ deficiency.
人体（以及实验动物）受到直接且极其危险的影响时，会自动开始呈现出一种独特的昏睡综合征形式的古老低体温保护反应。然而，低体温状态在进化上与高级脑结构格格不入。新旧调节系统之间会出现功能冲突。调节病理的加剧形成了具有危险病理成分和阶段过程的整个典型刻板休克综合征。然而，休克维持了机体的生命力，并使其在初始崩溃的破坏性影响后免于立即死亡。结果取决于抗休克治疗的充分性、机体生物储备的保存程度以及器官功能不全的严重程度。
