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近乎致命的空气栓塞:纤维蛋白鞘作为空气进入的通道。

Near-fatal air embolism: fibrin sheath as the portal of air entry.

作者信息

Roberts Shawn, Johnson Michelle, Davies Scott

机构信息

Department of Surgery, Hennepin County Medical Center, Minneapolis, MN 55415, USA.

出版信息

South Med J. 2003 Oct;96(10):1036-8. doi: 10.1097/01.SMJ.0000051067.20708.5F.

Abstract

Complications of central venous catheters occur in less than 1% of all insertions. Of these, pulmonary air embolism is a rare though often fatal complication. Possible mechanisms include opening of the line to the atmosphere during use and poor technique during insertion or removal. There has also been speculation that the presence of a fibrin sheath after removal might be a mechanism for air entry. We present a case of a near-fatal pulmonary air embolus with documentation of air in the pulmonary outflow trunk and a residual air-filled fibrin tract seen on computed tomography.

摘要

中心静脉导管并发症在所有置管操作中发生率不到1%。其中,肺空气栓塞虽罕见但常致命。可能的机制包括使用过程中导管与大气相通以及置管或拔管技术不佳。也有人推测拔管后纤维蛋白鞘的存在可能是空气进入的一种机制。我们报告一例近乎致命的肺空气栓塞病例,计算机断层扫描显示肺动脉干有空气且存在残留的充满空气的纤维蛋白通道。

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