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肾衰竭中大量软组织钙化导致伸肌腱功能完全丧失。

Massive soft tissue calcification causing complete loss of extensor tendon function in renal failure.

作者信息

Schenkier S L, Gertner E

机构信息

Section of Rheumatology, St. Paul-Ramsey Medical Center, MN 55101.

出版信息

J Rheumatol. 1992 Oct;19(10):1640-2.

PMID:1464882
Abstract

Extraskeletal soft tissue calcifications occur commonly in patients with uremia receiving dialysis. Rarely, a large tumoral calcinosis-like mass may develop. A patient receiving chronic ambulatory peritoneal dialysis for only 7 months developed a tumoral calcinosis-like mass that encased the extensor tendons of his wrist with loss of extensor tendon function, initially suggesting extensor tendon rupture. Surgical debridement restored tendon function. Tumoral calcinosis-like lesions are uncommon, but may cause limitation of joint movement, pain or ulceration through the skin. Measures aimed at controlling factors contributing to soft tissue calcification should be undertaken in any event whether surgery is required or not.

摘要

骨骼外软组织钙化常见于接受透析的尿毒症患者。罕见的情况下,可能会形成巨大的肿瘤样钙质沉着肿块。一名仅接受了7个月持续性非卧床腹膜透析的患者出现了肿瘤样钙质沉着肿块,该肿块包裹了其腕部的伸肌腱,导致伸肌腱功能丧失,最初提示伸肌腱断裂。手术清创恢复了肌腱功能。肿瘤样钙质沉着病变并不常见,但可能会导致关节活动受限、疼痛或皮肤溃疡。无论是否需要手术,都应采取措施控制导致软组织钙化的因素。

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