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Bradykinin and asthma.

作者信息

Barnes P J

机构信息

Department of Thoracic Medicine, National Heart and Lung Institute, London.

出版信息

Thorax. 1992 Nov;47(11):979-83. doi: 10.1136/thx.47.11.979.

DOI:10.1136/thx.47.11.979
PMID:1465760
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC464122/
Abstract
摘要

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本文引用的文献

1
Bradykinin stimulates afferent vagal C-fibers in intrapulmonary airways of dogs.缓激肽刺激犬肺内气道的传入迷走神经C纤维。
J Appl Physiol Respir Environ Exerc Physiol. 1980 Mar;48(3):511-7. doi: 10.1152/jappl.1980.48.3.511.
2
Pharmacology of bradykinin and related kinins.缓激肽及相关激肽的药理学
Pharmacol Rev. 1980 Mar;32(1):1-46.
3
In vivo and in vitro effect of bradykinin on bronchial motor tone in normal subjects and patients with airways obstruction.缓激肽对正常受试者和气道阻塞患者支气管运动张力的体内及体外效应。
Respiration. 1973;30(4):378-88. doi: 10.1159/000193051.
4
Competitive antagonists of bradykinin.缓激肽的竞争性拮抗剂。
Peptides. 1985 Mar-Apr;6(2):161-4. doi: 10.1016/0196-9781(85)90033-6.
5
Bradykinin stimulates Cl secretion and prostaglandin E2 release by canine tracheal epithelium.缓激肽刺激犬气管上皮细胞的氯离子分泌和前列腺素E2释放。
Am J Physiol. 1985 Jan;248(1 Pt 2):F48-55. doi: 10.1152/ajprenal.1985.248.1.F48.
6
The impact of passive smoking on emergency room visits of urban children with asthma.被动吸烟对城市哮喘儿童急诊就诊的影响。
Am Rev Respir Dis. 1987 Mar;135(3):567-72. doi: 10.1164/arrd.1987.135.3.567.
7
Bradykinin-induced bronchoconstriction in humans. Mode of action.缓激肽诱导的人类支气管收缩。作用模式。
Am Rev Respir Dis. 1987 Jan;135(1):176-80. doi: 10.1164/arrd.1987.135.1.176.
8
Detection of tissue kallikrein in the bronchoalveolar lavage fluid of asthmatic subjects.哮喘患者支气管肺泡灌洗液中组织激肽释放酶的检测
J Clin Invest. 1987 Jan;79(1):188-97. doi: 10.1172/JCI112782.
9
Kinin formation: mechanisms and role in inflammatory disorders.激肽形成:炎症性疾病中的机制及作用
Annu Rev Immunol. 1988;6:49-83. doi: 10.1146/annurev.iy.06.040188.000405.
10
Identification of human lung mast cell kininogenase as tryptase and relevance of tryptase kininogenase activity.鉴定人肺肥大细胞激肽原酶为类胰蛋白酶及其类胰蛋白酶激肽原酶活性的相关性。
Biochem Pharmacol. 1988 Apr 15;37(8):1473-80. doi: 10.1016/0006-2952(88)90008-1.