Tada Hiroshi, Naito Shigeto, Meguro Kentaro, Nogami Akihiko, Taniguchi Koichi
Division of Cardiology, Gunma Prefectural Cardiovascular Center, Kameizumi, Maebashi, Gunma, Japan.
Pacing Clin Electrophysiol. 2004 Feb;27(2):252-5. doi: 10.1111/j.1540-8159.2004.00421.x.
We describe a patient with drug-resistant paroxysmal atrial fibrillation who underwent radiofrequency catheter ablation of tachycardia originating from the superior vena cava (SVC). A continuous and rapid tachycardia conducted to the atrium with evidence of exit block and drove atrial fibrillation (AF) and atrial tachycardia. Neither AF nor atrial tachycardia could be induced after SVC isolation, and nor were they observed during follow-up. We conclude that continuous and rapid tachycardia originating from the SVC can act as a driver as well as an initiator of AF.
我们描述了一名患有耐药性阵发性心房颤动的患者，该患者接受了源于上腔静脉（SVC）的心动过速的射频导管消融术。一种连续且快速的心动过速传导至心房，有传出阻滞的证据，并引发了心房颤动（AF）和房性心动过速。在隔离上腔静脉后，既不能诱发AF也不能诱发房性心动过速，在随访期间也未观察到它们。我们得出结论，源于上腔静脉的连续且快速的心动过速可作为AF的驱动因素以及起始因素。
