Surawicz B, Saito S
Am J Cardiol. 1978 May 1;41(5):943-51. doi: 10.1016/0002-9149(78)90738-5.
This review consists of two parts: (1) discussion of the electrophysiologic mechanisms that are believed to produce ventricular repolarization changes during the electrocardiographic stress test, and (2) clinical assessment of the electrocardiographic changes with stress in patients with an abnormal electrocardiogram at rest. In the first part, the mechanisms of S-T segment elevation, S-T segment depression, T wave changes and linked S-T and T wave changes are reviewed. In the second part, all electrocardiographic abnormalities at rest are grouped into four categories: (1) changes that mask the manifestations of ischemia, (2) changes that stimulate or exaggerate the manifestations of ischemia, (3) changes that have no important effect on the manifestations of ischemia, and (4) changes that reproduce the patterns of acute myocardial infarction after an apparent healing. The reported studies of electrocardiographic stress testing in patients who have abnormal electrocardiogram at rest are summarized.
（1）讨论在心电图负荷试验期间被认为会导致心室复极改变的电生理机制，以及（2）对静息心电图异常患者负荷时心电图变化的临床评估。在第一部分中，回顾了ST段抬高、ST段压低、T波改变以及相关的ST段和T波改变的机制。在第二部分中，所有静息心电图异常被分为四类：（1）掩盖缺血表现的改变，（2）刺激或夸大缺血表现的改变，（3）对缺血表现无重要影响的改变，以及（4）在明显愈合后重现急性心肌梗死模式的改变。总结了关于静息心电图异常患者心电图负荷试验的已报道研究。
