Tsuchido T, Takeuchi A, Takano M
Department of Biotechnology, Kansai University, Japan.
J Appl Bacteriol. 1992 Dec;73(6):531-4. doi: 10.1111/j.1365-2672.1992.tb05017.x.
Escherichia coli cells exposed to a sublethal heat treatment at 55 degrees C for 15s synthesized lipopolysaccharide during their recovery period after heat stress. As chloramphenicol at least partly inhibited the synthesis of lipopolysaccharide, it is suggested that its synthesis might be in part due to the lipopolysaccharide-synthesizing enzymes produced de novo. The results obtained coincided with our previous finding that the permeability barrier function was repaired by heat-stressed cells.
暴露于55摄氏度亚致死热处理15秒的大肠杆菌细胞在热应激后的恢复期合成了脂多糖。由于氯霉素至少部分抑制了脂多糖的合成，表明其合成可能部分归因于重新产生的脂多糖合成酶。所得结果与我们之前发现热应激细胞修复通透性屏障功能相吻合。
