Liu Xuejun, Lu Ping, Chen Hanqi, Zhang Ziming, Tong Shuwen, Liu Jing, Gao Xiuying
Department of Anatomy, North China Coal Medical College, Tangshan 063000.
Lin Chuang Er Bi Yan Hou Ke Za Zhi. 2003 Nov;17(11):654-6.
In order to provide morphological evidences for olfactory defect cased by olfactory bulb, olfactory tract ischemia, the origins, numbers, distributions and pathological changes about olfactory bulb, olfactory tract arteries were studied.
The distributions and pathological changes of olfactory bulb, olfactory tract arteries in 80 sides of adult brain specimens were observed with operation microscope, among which the nourishing arteries and nerve of olfactory tract in two sides aged from 60 to 70 were observed pathohistologically.
The blood supply of olfactory bulb, olfactory tract comes mainly form artery cerebral and posterior communicainy. 10% of these 60 sides arteries were of multi-branch. 86.3% of them had atheroselerosis. 25.0% of the olfactory bulb and olfactory tract artery were blocked or narrow, and pathological changes in olfactory nerve such as atrophy were observed.
Excluding other diseases, the olfactory defect in different degree of patients over 60 years old were caused possibly by atheroselerosis of the arteries at base of the brain.
为嗅球、嗅束缺血所致嗅觉障碍提供形态学依据,研究嗅球、嗅束动脉的起源、数目、分布及病理变化。
在手术显微镜下观察80侧成人大脑标本中嗅球、嗅束动脉的分布及病理变化,其中对2例60~70岁老人双侧嗅束的营养动脉及神经进行病理组织学观察。
嗅球、嗅束的血液供应主要来自大脑前动脉及后交通动脉。60侧动脉中10%为多支型,86.3%有动脉粥样硬化。25.0%的嗅球、嗅束动脉发生阻塞或狭窄,并观察到嗅神经萎缩等病理变化。
排除其他疾病,60岁以上患者不同程度的嗅觉障碍可能是由脑底动脉粥样硬化所致。