Uchino Akira, Sawada Akihiro, Takase Yukinori, Nojiri Jun-ichi, Kudo Sho
Department of Radiology, Saga Medical School Nabeshima, Saga 849-8501, Japan.
Radiat Med. 2004 Jan-Feb;22(1):37-41.
To report magnetic resonance (MR) imaging findings of wallerian degeneration of the pontocerebellar tract secondary to a pontine infarction.
We retrospectively reviewed cranial MR images obtained during the past seven years in our institution and selected those from patients with a chronic stage of pontine infarction and a hyperintense lesion at the central portion of the middle cerebellar peduncle on T2-weighted images.
In three patients with a ventromedial pontine infarction, we found a symmetrical hyperintense lesion at the central portion of the middle cerebellar peduncle bilaterally on T2-weighted MR images in the chronic stage. In another patient with a ventrolateral pontine infarction, we found such a lesion at the ipsilateral middle cerebellar peduncle.
Because the middle cerebellar peduncle carries afferent fibers from the contralateral basis pontis to the cerebellar cortex, these middle cerebellar peduncular lesions are regarded as wallerian degeneration. This secondary degeneration should not be misinterpreted as a newly developed infarction or other disease.
报告桥脑梗死继发桥小脑束华勒氏变性的磁共振（MR）成像表现。
我们回顾性分析了本机构过去七年中获得的头颅MR图像，选取了处于桥脑梗死慢性期且在T2加权图像上小脑桥脑脚中部有高信号病变的患者的图像。
在3例桥脑腹内侧梗死患者中，我们发现在慢性期T2加权MR图像上双侧小脑桥脑脚中部有对称的高信号病变。在另一例桥脑腹外侧梗死患者中，我们在同侧小脑桥脑脚发现了这样一个病变。
由于小脑桥脑脚携带来自对侧脑桥基底部至小脑皮质的传入纤维，这些小脑桥脑脚病变被视为华勒氏变性。这种继发性变性不应被误解为新发生的梗死或其他疾病。
