Macêdo S B, Ferreira L R, Perazzo F F, Carvalho J C
Faculdade de Odontologia, Pontificia Universidade Católica de Brasília, Brasília, Brazil.
Homeopathy. 2004 Apr;93(2):84-7. doi: 10.1016/j.homp.2004.02.006.
The anti-inflammatory effect of Arnica montana 6cH was evaluated using acute and chronic inflammation models. In the acute, model, carrageenin-induced rat paw oedema, the group treated with Arnica montana 6cH showed 30% inhibition compared to control (P < 0.05). Treatment with Arnica 6cH, 30 min prior to carrageenin, did not produce any inhibition of the inflammatory process. In the chronic model, Nystatin-induced oedema, the group treated 3 days previously with Arnica montana 6cH had reduced inflammation 6 h after the inflammatory agent was applied (P < 0.05). When treatment was given 6 h after Nystatin treatment, there was no significant inhibitory effect. In a model based on histamine-induced increase of vascular permeability, pretreatment with Arnica montana 6cH blocked the action of histamine in increasing vascular permeability.
采用急性和慢性炎症模型评估了山金车6cH的抗炎作用。在急性模型中，对角叉菜胶诱导的大鼠足爪水肿，与对照组相比，用山金车6cH治疗的组显示出30%的抑制作用（P<0.05）。在角叉菜胶给药前30分钟用山金车6cH治疗，未对炎症过程产生任何抑制作用。在慢性模型中，对制霉菌素诱导的水肿，预先用山金车6cH治疗3天的组在应用炎症剂后6小时炎症减轻（P<0.05）。当在制霉菌素治疗后6小时给予治疗时，没有显著的抑制作用。在基于组胺诱导的血管通透性增加的模型中，用山金车6cH预处理可阻断组胺增加血管通透性的作用。
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