Pineda Pompa Luis Ramón, Barrera-Ramírez Carlos Felipe, Martínez-Valdez Jesús, Rodríguez Pedro Domínguez, Guzmán Carlos E
División Cardiovascular, Servicios de Cardiología Intervencionista, Terapia Intensiva y Radiología, Centro Hospitalario la Concepción, Saltillo, México.
Rev Port Cardiol. 2004 Apr;23(4):561-8.
Acute noncardiogenic pulmonary edema and catecholamine-induced cardiomyopathy as the first presentations of pheochromocytoma are uncommon events, but usually rapidly fatal. A 36-year-old man presented acute pulmonary edema in a setting of hypertensive emergency after arthroscopy, later developing catecholamine-induced cardiotoxicity mimicking an acute myocardial infarction, with elevation of cardiac damage markers, normal coronary arteries, and with full recovery from electrical abnormalities. Magnetic resonance imaging revealed a right adrenal mass. Elevated levels of catecholamines and metanephrines, and a positive 131I-metaiodobenzylguanidine scan confirmed a pheochromocytoma. Once the patient had been hemodynamically stabilized, he was successfully operated.
急性非心源性肺水肿和儿茶酚胺诱导的心肌病作为嗜铬细胞瘤的首发表现并不常见，但通常迅速致命。一名36岁男性在关节镜检查后出现高血压急症，继而发生急性肺水肿，随后出现类似急性心肌梗死的儿茶酚胺诱导的心脏毒性，心脏损伤标志物升高，冠状动脉正常，且电异常完全恢复。磁共振成像显示右侧肾上腺肿块。儿茶酚胺和甲氧基肾上腺素水平升高，以及131I-间碘苄胍扫描阳性证实为嗜铬细胞瘤。患者血流动力学稳定后，成功接受了手术。
