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Genetic pathways to melanoma tumorigenesis.黑色素瘤肿瘤发生的遗传途径。
J Clin Pathol. 2004 Aug;57(8):797-801. doi: 10.1136/jcp.2003.015800.
2
Tumor progression in the human melanocytic system.人类黑素细胞系统中的肿瘤进展。
Anticancer Res. 1989 Jul-Aug;9(4):865-72.
3
Allelotypes of primary cutaneous melanoma and benign melanocytic nevi.原发性皮肤黑色素瘤和良性黑素细胞痣的等位基因型。
Cancer Res. 1996 Feb 1;56(3):589-93.
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X inactivation, DNA deletion, and microsatellite instability in common acquired melanocytic nevi.常见获得性黑素细胞痣中的X染色体失活、DNA缺失和微卫星不稳定性
Clin Cancer Res. 2001 Dec;7(12):4054-9.
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Use of microgenomic technology for analysis of alterations in DNA copy number and gene expression in malignant melanoma.利用微基因组技术分析恶性黑色素瘤中DNA拷贝数改变和基因表达情况。
Clin Exp Immunol. 1997 Jan;107 Suppl 1:33-40.
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Novel genes associated with malignant melanoma but not benign melanocytic lesions.与恶性黑色素瘤相关但与良性黑素细胞病变无关的新基因。
Clin Cancer Res. 2005 Oct 15;11(20):7234-42. doi: 10.1158/1078-0432.CCR-05-0683.
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Molecular alterations at chromosome 9p21 in melanocytic naevi and melanoma.黑素细胞痣和黑色素瘤中9号染色体短臂21区的分子改变。
Br J Dermatol. 2008 Feb;158(2):243-50. doi: 10.1111/j.1365-2133.2007.08310.x. Epub 2007 Nov 19.
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A defined region of loss of heterozygosity at 11q23 in cutaneous malignant melanoma.皮肤恶性黑色素瘤中11q23处杂合性缺失的一个明确区域。
Cancer Res. 1995 Jun 15;55(12):2494-6.
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Concordant loss of heterozygosity of DNA repair gene, hOGG1, in melanoma in situ and atypical melanocytic hyperplasia.原位黑色素瘤和非典型黑素细胞增生中DNA修复基因hOGG1的杂合性一致缺失。
J Cutan Pathol. 2008 Jun;35(6):525-31. doi: 10.1111/j.1600-0560.2007.00865.x. Epub 2008 Feb 28.
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Analysis of losses of heterozygosity of the candidate tumour suppressor gene DMBT1 in melanoma resection specimens.黑色素瘤切除标本中候选抑癌基因DMBT1的杂合性缺失分析。
Oncology. 2002;63(2):166-72. doi: 10.1159/000063802.
引用本文的文献
1
Cutaneous Malignant Melanoma Presenting as an Isolated Splenic Metastasis: An Update.以孤立性脾转移为表现的皮肤恶性黑色素瘤：最新进展
World J Oncol. 2023 Feb;14(1):21-25. doi: 10.14740/wjon1567. Epub 2023 Feb 26.
2
The immune-related role of beta-2-microglobulin in melanoma.β2微球蛋白在黑色素瘤中的免疫相关作用。
Front Oncol. 2022 Aug 16;12:944722. doi: 10.3389/fonc.2022.944722. eCollection 2022.
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1,2-Dihydroxyxanthone: Effect on A375-C5 Melanoma Cell Growth Associated with Interference with THP-1 Human Macrophage Activity.1,2-二羟基呫吨酮：对A375-C5黑色素瘤细胞生长的影响及与干扰THP-1人巨噬细胞活性的关联
Pharmaceuticals (Basel). 2019 Jun 4;12(2):85. doi: 10.3390/ph12020085.
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Exploring major signaling cascades in melanomagenesis: a rationale route for targetted skin cancer therapy.探讨黑素瘤发生中的主要信号级联反应：靶向皮肤癌治疗的合理途径。
Biosci Rep. 2018 Oct 2;38(5). doi: 10.1042/BSR20180511. Print 2018 Oct 31.
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Microradiopharmaceutical for Metastatic Melanoma.用于转移性黑色素瘤的微放射性药物。
Pharm Res. 2017 Dec;34(12):2922-2930. doi: 10.1007/s11095-017-2275-3. Epub 2017 Oct 23.
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Quantitative comparison of the spreading and invasion of radial growth phase and metastatic melanoma cells in a three-dimensional human skin equivalent model.在三维人体皮肤等效模型中对径向生长阶段和转移性黑色素瘤细胞的扩散与侵袭进行定量比较。
PeerJ. 2017 Sep 5;5:e3754. doi: 10.7717/peerj.3754. eCollection 2017.
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Retrospective multicenter evaluation of patients diagnosed with mucosal melanoma: a study of Anatolian Society of Medical Oncology.黏膜黑色素瘤确诊患者的回顾性多中心评估：一项安纳托利亚医学肿瘤学会的研究
Tumour Biol. 2016 Sep;37(9):12033-12038. doi: 10.1007/s13277-016-5076-0. Epub 2016 May 12.
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Deficient mismatch repair: Read all about it (Review).错配修复缺陷：全面了解（综述）。
Int J Oncol. 2015 Oct;47(4):1189-202. doi: 10.3892/ijo.2015.3119. Epub 2015 Aug 12.
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Mutational status of naevus-associated melanomas.痣相关黑色素瘤的突变状态。
Br J Dermatol. 2015 Sep;173(3):671-80. doi: 10.1111/bjd.13829. Epub 2015 Jun 19.
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Isolated limb infusion as a model to test new agents to treat metastatic melanoma.孤立肢体灌注作为一种模型来测试治疗转移性黑色素瘤的新药物。
J Surg Oncol. 2014 Mar;109(4):357-65. doi: 10.1002/jso.23502. Epub 2013 Nov 20.
本文引用的文献
1
hMLH1 and hMSH2 gene mutations are present in radial growth-phase cutaneous malignant melanoma cell lines and can be induced further by ultraviolet-B irradiation.hMLH1和hMSH2基因突变存在于放射状生长期皮肤恶性黑色素瘤细胞系中，并且可通过紫外线B照射进一步诱导产生。
Exp Dermatol. 2003 Dec;12(6):872-5. doi: 10.1111/j.0906-6705.2003.00104.x.
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Progression in cutaneous malignant melanoma is associated with distinct expression profiles: a tissue microarray-based study.皮肤恶性黑色素瘤的进展与不同的表达谱相关：一项基于组织微阵列的研究。
Am J Pathol. 2004 Jan;164(1):193-203. doi: 10.1016/s0002-9440(10)63110-0.
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Identification of novel deletion Loci at 1p36 and 9p22-21 in melanocytic dysplastic nevi and cutaneous malignant melanomas.在黑素细胞发育异常痣和皮肤恶性黑色素瘤中1p36以及9p22 - 21区域新型缺失位点的鉴定
Arch Dermatol. 2003 Jun;139(6):816-7. doi: 10.1001/archderm.139.6.816.
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DNA microarrays and likelihood ratio bioinformatic methods: discovery of human melanocyte biomarkers.DNA微阵列与似然比生物信息学方法：人类黑素细胞生物标志物的发现
Pigment Cell Res. 2003 Jun;16(3):245-53. doi: 10.1034/j.1600-0749.2003.00036.x.
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Assessment of genetic instability in melanocytic skin lesions through microsatellite analysis of benign naevi, dysplastic naevi, and primary melanomas and their metastases.通过对良性痣、发育异常痣、原发性黑色素瘤及其转移灶进行微卫星分析，评估黑素细胞性皮肤病变中的基因不稳定性。
Melanoma Res. 2003 Apr;13(2):167-70. doi: 10.1097/00008390-200304000-00009.
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Genotypic analysis of primary and metastatic cutaneous melanoma.原发性和转移性皮肤黑色素瘤的基因分型分析。
Cancer Genet Cytogenet. 2003 Jan 1;140(1):37-44. doi: 10.1016/s0165-4608(02)00651-9.
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Gene expression profiling of melanocytic lesions.黑素细胞性病变的基因表达谱分析。
Am J Dermatopathol. 2003 Feb;25(1):6-11. doi: 10.1097/00000372-200302000-00002.
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Microsatellite analysis in cutaneous malignant melanoma.皮肤恶性黑色素瘤的微卫星分析
Melanoma Res. 2002 Dec;12(6):577-84. doi: 10.1097/00008390-200212000-00007.
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Loss of heterozygosity, microsatellite instability, and mismatch repair protein alterations in the radial growth phase of cutaneous malignant melanomas.皮肤恶性黑色素瘤放射状生长期的杂合性缺失、微卫星不稳定性及错配修复蛋白改变
Mol Carcinog. 2002 May;34(1):35-44. doi: 10.1002/mc.10047.
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Microsatellite instability and its relevance to cutaneous tumorigenesis.微卫星不稳定性及其与皮肤肿瘤发生的相关性。
J Cutan Pathol. 2002 May;29(5):257-67. doi: 10.1034/j.1600-0560.2002.290501.x.
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