ATP-induced inhibition of mitochondrial ATPase by oligomycin.
作者信息
Drobinskaya I E, Kozlov I A, Skulachev V P
出版信息
FEBS Lett. 1978 Dec 1;96(1):111-4. doi: 10.1016/0014-5793(78)81073-4.
PMID:153239
Abstract
摘要
相似文献
1
ATP-induced inhibition of mitochondrial ATPase by oligomycin.寡霉素对ATP诱导的线粒体ATP酶的抑制作用。
FEBS Lett. 1978 Dec 1;96(1):111-4. doi: 10.1016/0014-5793(78)81073-4.
2
Natural protein ATPase inhibitor from Candida utilis mitochondria. Binding properties of the radiolabeled inhibitor.来自产朊假丝酵母线粒体的天然蛋白质ATP酶抑制剂。放射性标记抑制剂的结合特性。
FEBS Lett. 1977 Dec 1;84(1):129-34. doi: 10.1016/0014-5793(77)81072-7.
3
[Esterase activity of the mitochondria oligomycin-sensitive ATPase complex].[线粒体寡霉素敏感ATP酶复合体的酯酶活性]
Biokhimiia. 1978 Nov;43(11):2058-63.
4
Kinetic studies of beef heart mitochondrial adenosine triphosphatase: interaction of the inhibitor protein and adenosine triphosphate analogues.牛心线粒体三磷酸腺苷酶的动力学研究：抑制蛋白与三磷酸腺苷类似物的相互作用
Biochemistry. 1981 Mar 17;20(6):1592-8. doi: 10.1021/bi00509a028.
5
Control of proton conduction by the H+ -ATPase in the inner mitochondrial membrane.
Eur J Biochem. 1978 Dec;92(2):545-51. doi: 10.1111/j.1432-1033.1978.tb12776.x.
6
Photoaffinity cross-linking of oligomycin-sensitive ATPase from beef heart mitochondria by 3'-arylazido-8-azido ATP.用3'-芳基叠氮基-8-叠氮基ATP对牛心线粒体寡霉素敏感的ATP酶进行光亲和交联。
Biochem Biophys Res Commun. 1983 Mar 16;111(2):732-9. doi: 10.1016/0006-291x(83)90366-2.
7
Reconstituted mitochondrial oligomycin-sensitive ATPase (F0F1) with intermediate Pi in equilibrium HOH exchange but no Pi in equilibrium ATP exchange activity.重构的线粒体寡霉素敏感ATP酶（F0F1），在平衡态水交换中有中等水平的无机磷酸（Pi），但在平衡态ATP交换活性中无Pi。
FEBS Lett. 1977 Dec 15;84(2):283-6. doi: 10.1016/0014-5793(77)80707-2.
8
Oligomycin-sensitive ATPase from beef heart mitochondria: reaction with 2-mercaptopropionylglycine.
FEBS Lett. 1978 Oct 15;94(2):223-7. doi: 10.1016/0014-5793(78)80942-9.
9
Coupling factor B is a component of the Fo proton channel of mitochondrial H+-ATPase.偶联因子B是线粒体H⁺-ATP酶Fo质子通道的一个组成部分。
J Biol Chem. 1987 Mar 5;262(7):3007-10.
10
Mitochondrial ATPase inactivation by interaction with its substrate.
Arch Biochem Biophys. 1982 Apr 15;215(1):40-6. doi: 10.1016/0003-9861(82)90276-4.
引用本文的文献
1
Impaired nuclear Nrf2 translocation undermines the oxidative stress response in Friedreich ataxia.核Nrf2易位受损会破坏弗里德赖希共济失调中的氧化应激反应。
PLoS One. 2009;4(1):e4253. doi: 10.1371/journal.pone.0004253. Epub 2009 Jan 22.
Zotero 插件