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Paget disease of bone.
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Paget's disease of bone is characterized by excessive bone resorption coupled with excessive and disorganized bone formation.
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[What is Paget's disease?].
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Osteoclasts formed by measles virus-infected osteoclast precursors from hCD46 transgenic mice express characteristics of pagetic osteoclasts.
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[Quantitative histology and dynamics of pagetic bone (author's transl)].
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Etiology of Paget's disease and osteoclast abnormalities.
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Paget's disease of bone.
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Epidemiology and pathology of Paget's disease of bone - a review.
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Therapeutic Efficacy of Medicinal Plants with Allopathic Medicine in Musculoskeletal Diseases.
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Unnatural helical peptidic foldamers as protein segment mimics.
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The Ying and Yang of Sphingosine-1-Phosphate Signalling within the Bone.
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Molecular test of Paget's disease of bone in families not linked to gene mutations.
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Oligoarginine-Conjugated Peptide Foldamers Inhibiting Vitamin D Receptor-Mediated Transcription.
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Paget's disease (Osteitis deformans) and heredity.
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Role of TAFII-17, a VDR binding protein, in the increased osteoclast formation in Paget's Disease.
J Bone Miner Res. 2004 Jul;19(7):1154-64. doi: 10.1359/JBMR.040312. Epub 2004 Mar 15.
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Novel UBA domain mutations of SQSTM1 in Paget's disease of bone: genotype phenotype correlation, functional analysis, and structural consequences.
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The role of cathepsin K in normal bone resorption.
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The atypical PKC-interacting protein p62 is an important mediator of RANK-activated osteoclastogenesis.
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Three novel mutations in SQSTM1 identified in familial Paget's disease of bone.
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Fibroblast growth factor signaling controlling osteoblast differentiation.
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Controlling mesenchymal stem cell differentiation by TGFBeta family members.
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New insights into the molecular mechanisms of action of bisphosphonates.
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Phenotypic characterization of early onset Paget's disease of bone caused by a 27-bp duplication in the TNFRSF11A gene.
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