Kissel P, Youmans J R
Sierra Vista Regional Medical Center, San Luis Obispo, California.
J Spinal Disord. 1992 Mar;5(1):104-7. doi: 10.1097/00002517-199203000-00014.
Degenerative changes in the cervical spine can produce osteophytes and other hypertrophic abnormalities. Asymptomatic osteophytes of the anterior margins of the cervical vertebrae may occur in 20-30% of the population. Occasionally, dysphagia or dysphonia may be caused by such cervical osteophytes pressing against the esophagus or trachea. Recently, the authors treated a patient with posttraumatic dysphagia and dysphonia secondary to osteophytic spurring of the anterior cervical spine. This 43-year-old man presented 2 years after sustaining a flexion/extension soft tissue injury to his cervical spine. Radiographic studies depict the progression of his osteophyte growth, which resulted in surgical intervention to relieve his inability to swallow solid foods. One year follow-up studies demonstrate normal alignment and no instability. A search of the literature revealed approximately 75 previously reported cases of anterior osteophyte-induced dysphagia, with the majority secondary to diffuse idiopathic skeletal hyperostosis. The literature briefly mentions trauma as a possible etiology of anterior osteophytosis; however, our case is unique, as it documents the time course and progression of the pathologic process.
颈椎的退行性变可产生骨赘及其他肥大性异常。颈椎椎体前缘无症状性骨赘在20% - 30%的人群中可能出现。偶尔，此类颈椎骨赘压迫食管或气管可导致吞咽困难或发音障碍。最近，作者治疗了一名因颈椎前路骨赘增生继发创伤后吞咽困难和发音障碍的患者。这名43岁男性在颈椎遭受屈伸性软组织损伤2年后就诊。影像学研究显示了其骨赘生长的进展情况，这导致了手术干预以缓解其无法吞咽固体食物的状况。一年的随访研究显示排列正常且无不稳定情况。文献检索发现约有75例先前报道的前路骨赘引起吞咽困难的病例，其中大多数继发于弥漫性特发性骨肥厚。文献简要提及创伤是前路骨赘形成的可能病因；然而，我们的病例是独特的，因为它记录了病理过程的时间进程和进展情况。
