Sternick Eduardo Back, Fagundes Márcio L, Cruz Fernando E S, Timmermans Carl, Sosa Eduardo A, Rodriguez Luz-Maria, Gerken Luiz M, Scanavacca Maurício I, Wellens Hein J J
Biocor Instituto, Nova Lima, Brazil.
J Cardiovasc Electrophysiol. 2005 Feb;16(2):127-34. doi: 10.1046/j.1540-8167.2004.40508.x.
Short A-V manheim fiber.
A short atrioventricular decrementally conducting accessory pathway is an uncommon variant of preexcitation. Available data from small series suggest that their decremental properties might not be caused by A-V nodal-like tissue.
We compared clinical, electrocardiographic and electrophysiologic parameters in two groups of patients: 8 patients with a short A-V Mahaim pathway (Group A), and 33 patients with atriofascicular pathways (Group B). Radiofrequency catheter ablation was carried out guided by activation mapping at the annulus in Group A patients and targeting the "M" potential in Group B patients.
After ablation of all associated rapidly conducting bypass tracts, 7 of the 8 Group A patients showed clear preexcitation. In only 1 of 8 patients the short A-V Mahaim fiber was actively engaged in a reentrant tachycardia circuit. During radiofrequency catheter ablation an automatic rhythm occurred in 4 of 8 patients. Intravenous adenosine caused conduction a block in the Mahaim fiber in 3 of the 5 patients tested. In group B, no patient showed clear preexcitation (P<00001) while 72% had a minimal preexcitation pattern. Twenty-nine of the 33 patients had a circus movement tachycardia with AV conduction over the atriofascicular fiber. During radiofrequency catheter ablation 30 of 33 patients showed accessory pathway automaticity. Adenosine caused transient block at the atriofascicular pathway in 11 (92%) of the 12 patients tested.
While short decrementally conducting right-sided accessory pathways show a typical ECG pattern different from atriofascicular pathways, their electrophysiologic properties do not seem to be uniform. Those pathways can be successfully interrupted by catheter ablation.
短房室旁道纤维。
短的房室递减传导旁路是预激综合征的一种罕见变异类型。来自小样本系列研究的现有数据表明,其递减特性可能并非由类似房室结的组织引起。
我们比较了两组患者的临床、心电图和电生理参数:8例有短房室Mahaim旁道的患者(A组)和33例有房束旁道的患者(B组)。A组患者在瓣环处激动标测引导下行射频导管消融,B组患者以“M”电位为靶点进行消融。
在消融所有相关的快速传导旁路后,8例A组患者中有7例仍有明显预激。8例患者中仅1例短房室Mahaim纤维积极参与折返性心动过速环路。在射频导管消融期间,8例患者中有4例出现自律性心律。静脉注射腺苷使5例接受测试的患者中的3例Mahaim纤维发生传导阻滞。在B组中,没有患者表现出明显预激(P<0.0001),而72%的患者有最小预激图形。33例患者中有29例通过房束纤维发生房室传导的 circus运动性心动过速。在射频导管消融期间,33例患者中有30例表现出旁路自律性。腺苷使12例接受测试的患者中的11例(92%)房束旁道发生短暂阻滞。
虽然短的递减传导右侧旁路表现出与房束旁道不同的典型心电图图形,但其电生理特性似乎并不一致。这些旁路可通过导管消融成功阻断。