Wilkenfeld C, Cohen M, Lansman S L, Courtney M, Dische M R, Pertsemlidis D, Krakoff L R
Department of Medicine, Mount Sinai School of Medicine, City University of New York, NY 10029.
J Heart Lung Transplant. 1992 Mar-Apr;11(2 Pt 1):363-6.
Dilated cardiomyopathy has been found in patients with pheochromocytoma. The cardiomyopathy and resultant congestive heart failure can be progressive and fatal, or reversible, if the source of the excess catecholamines is removed. Histologic and autopsy studies revealed a characteristic focal myocarditis and contraction band necrosis, indicating that the circulating catecholamine excess secreted by the pheochromocytoma is the cause of the cardiomyopathy.
在嗜铬细胞瘤患者中已发现扩张型心肌病。如果去除过量儿茶酚胺的来源,这种心肌病及由此导致的充血性心力衰竭可能会进展并致命,也可能是可逆的。组织学和尸检研究显示出特征性的局灶性心肌炎和收缩带坏死,表明嗜铬细胞瘤分泌的循环儿茶酚胺过量是心肌病的病因。