Janovská E, Samoĭlova K A, Vizdalová M
Tsitologiia. 1979 Jul;21(7):816-23.
A comparison has been made of sensitivity to far (254 nm), middle (300--315 nm) and near (315--400 nm) UV radiation of 12 strains of E. coli and 2 strains of B. subtilis differing in DNA dark repair (DR) capability. The mechanisms controlled by uvrA, uvrB, polA, recA, lon, and lexA genes are very effective in cells, irradiated by far and middle UV, but by 15--70% less effective in those irradiated by near UV. As the unirradiated bacteria poorly repair the near UV damaged phages (T7, lambda, SPPI), the low bacterial DR level after UV irradiation seems to be due to the unrepairable photoproduct formation in DNA.
对12株大肠杆菌和2株枯草芽孢杆菌在DNA暗修复（DR）能力上存在差异的菌株，进行了对远紫外线（254纳米）、中紫外线（300 - 315纳米）和近紫外线（315 - 400纳米）辐射敏感性的比较。由uvrA、uvrB、polA、recA、lon和lexA基因控制的机制在远紫外线和中紫外线照射的细胞中非常有效，但在近紫外线照射的细胞中效率要低15% - 70%。由于未照射的细菌对近紫外线损伤的噬菌体（T7、λ、SPPI）修复能力较差，紫外线照射后细菌的低DR水平似乎是由于DNA中形成了不可修复的光产物。
