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本文引用的文献
1
Blockade of Nogo-66, myelin-associated glycoprotein, and oligodendrocyte myelin glycoprotein by soluble Nogo-66 receptor promotes axonal sprouting and recovery after spinal injury.
J Neurosci. 2004 Nov 17;24(46):10511-20. doi: 10.1523/JNEUROSCI.2828-04.2004.
2
Nogo-66 receptor prevents raphespinal and rubrospinal axon regeneration and limits functional recovery from spinal cord injury.
Neuron. 2004 Oct 28;44(3):439-51. doi: 10.1016/j.neuron.2004.10.015.
3
Gait dynamics in trisomic mice: quantitative neurological traits of Down syndrome.
Physiol Behav. 2004 Sep 15;82(2-3):381-9. doi: 10.1016/j.physbeh.2004.04.006.
4
Nogo receptor antagonism promotes stroke recovery by enhancing axonal plasticity.
J Neurosci. 2004 Jul 7;24(27):6209-17. doi: 10.1523/JNEUROSCI.1643-04.2004.
5
Counteracting the Nogo receptor enhances optic nerve regeneration if retinal ganglion cells are in an active growth state.
J Neurosci. 2004 Feb 18;24(7):1646-51. doi: 10.1523/JNEUROSCI.5119-03.2004.
6
The injured spinal cord spontaneously forms a new intraspinal circuit in adult rats.
Nat Neurosci. 2004 Mar;7(3):269-77. doi: 10.1038/nn1195. Epub 2004 Feb 15.
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LINGO-1 is a component of the Nogo-66 receptor/p75 signaling complex.
Nat Neurosci. 2004 Mar;7(3):221-8. doi: 10.1038/nn1188. Epub 2004 Feb 15.
8
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EMBO J. 2003 Jul 1;22(13):3291-302. doi: 10.1093/emboj/cdg325.
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Nogo-C is sufficient to delay nerve regeneration.
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Neuroprotection by minocycline facilitates significant recovery from spinal cord injury in mice.
Brain. 2003 Jul;126(Pt 7):1628-37. doi: 10.1093/brain/awg178. Epub 2003 Jun 4.
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