Ogaard B, Rølla G
Faculty of Dentistry, University of Oslo, Blindern, Norway.
J Dent Res. 1992 Apr;71 Spec No:832-5. doi: 10.1177/002203459207100S08.
This paper presents the orthodontic banding model for vital teeth and the orthodontic in situ model for slabs of enamel, root surface, dentin, or other mineralized tissues such as shark enamel. The model for vital teeth is an in vivo model, since a crevice for plaque accumulation is created behind orthodontic bands on the buccal enamel surfaces of teeth in situ. Visible white-spot lesions are usually seen after a four-week banding period in the absence of fluoride. The microbiological flora developed behind the bands shows a similarity to that of natural caries. Microradiographic data show that the initial lesion is a softening of the enamel surface. Later, a subsurface lesion develops. A modification of the model has been developed for the use of slabs of mineralized tissues. In this model, slabs are mounted on a removable appliance. The slabs are covered with orthodontic banding material for plaque accumulation. Lesion development in enamel in the two model systems is almost identical. The benefit of the in vivo model is that caries development can be studied on vital teeth in young individuals. The model is independent of the patient's cooperation. No special diet is required, e.g., no sucrose rinsing. In the in situ model, slabs could be examined after one study period and then replaced for another period.
本文介绍了活髓牙的正畸带环模型以及牙釉质、牙根表面、牙本质或其他矿化组织（如鲨鱼牙釉质）板的正畸原位模型。活髓牙模型是一种体内模型，因为在原位牙齿颊侧牙釉质表面的正畸带环后方会形成菌斑积聚的缝隙。在无氟情况下，经过四周的带环期后通常会出现可见的白斑病变。带环后方形成的微生物菌群与自然龋的菌群相似。显微放射照相数据显示，初始病变是牙釉质表面软化。随后，会发展为表层下病变。已开发出该模型的一种改进形式，用于矿化组织板。在这个模型中，组织板安装在可移动矫治器上。组织板覆盖正畸带环材料以积聚菌斑。两个模型系统中牙釉质的病变发展几乎相同。体内模型的优点是可以在年轻个体的活髓牙上研究龋病发展。该模型不依赖患者的配合。不需要特殊饮食，例如，无需用蔗糖漱口。在原位模型中，经过一个研究期后可以检查组织板，然后更换组织板进行另一个研究期。
