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Increased biological activity of subchondral mineralized tissues underlies the progressive deterioration of articular cartilage in osteoarthritis.

作者信息

Burr David B

机构信息

Department of Anatomy and Cell Biology, Indiana University School of Medecine, Indianapolis, IN, USA.

出版信息

J Rheumatol. 2005 Jun;32(6):1156-8; discussion 1158-9.

PMID:15977355
Abstract
摘要

相似文献

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Increased biological activity of subchondral mineralized tissues underlies the progressive deterioration of articular cartilage in osteoarthritis.骨关节炎中,软骨下矿化组织生物活性增加是关节软骨进行性退变的基础。
J Rheumatol. 2005 Jun;32(6):1156-8; discussion 1158-9.
2
[The subchondral bone plaque].
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Biochemical markers for osteoarthritis: from the present to the future and back to the past.骨关节炎的生化标志物:从现在到未来,再回到过去。
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Subchondral bone in osteoarthritis: a biologic link with articular cartilage leading to abnormal remodeling.骨关节炎中的软骨下骨:与关节软骨的生物学联系导致异常重塑。
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[OVERLOAD of joints and its role in osteoarthritis. Towards understanding and preventing progression of primary osteoarthritis].[关节超负荷及其在骨关节炎中的作用。迈向对原发性骨关节炎进展的理解与预防]
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OVERLOAD of joints and its role in osteoarthritis : Towards understanding and preventing progression of primary osteoarthritis. English version.关节超负荷及其在骨关节炎中的作用:迈向对原发性骨关节炎进展的理解与预防。英文版
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Subchondral bone plate thickening precedes chondrocyte apoptosis and cartilage degradation in spontaneous animal models of osteoarthritis.在自发性骨关节炎动物模型中,软骨下骨板增厚先于软骨细胞凋亡和软骨降解。
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