Lee Bongsoo, Higgs Penelope I, Zusman David R, Cho Kyungyun
Section of Life Science, Hoseo University, Asan, Korea.
J Bacteriol. 2005 Jul;187(14):5029-31. doi: 10.1128/JB.187.14.5029-5031.2005.
The espC null mutation caused accelerated aggregation and formation of tiny fruiting bodies surrounded by spores, which were also observed in the espA mutant and in CsgA-overproducing cells in Myxococcus xanthus. In addition, the espC mutant appeared to produce larger amounts of the complementary C-signal than the wild-type strain. These findings suggest that EspC is involved in controlling the timing of fruiting body development in M. xanthus.
espC基因缺失突变导致聚集加速,并形成被孢子包围的微小子实体,在黄色粘球菌的espA突变体和过量表达CsgA的细胞中也观察到了这种情况。此外,espC突变体似乎比野生型菌株产生更多的互补C信号。这些发现表明,EspC参与控制黄色粘球菌子实体发育的时间。