Waldner H
Chirurgische Klinik Innenstadt, Ludwig-Maximilians-Universität, München, BRD.
Eur Surg Res. 1992;24 Suppl 1:62-7. doi: 10.1159/000129240.
Vascular mechanisms are not standard tools to induce acute pancreatitis. They are used for special purposes to investigate the pathophysiologic significance of circulatory changes in acute pancreatitis. Because of the rich collateral network, occlusion of the main pancreatic arteries induces no pathological change. Complete occlusion of the venous outflow induces hemorrhagic necrosis of the pancreas, disturbances of the microcirculation, edema and focal parenchymal necrosis as in acute pancreatitis. In the last model, pancreatic and systemic alterations can be examined.
血管机制并非诱导急性胰腺炎的标准手段。它们用于特殊目的，以研究急性胰腺炎循环变化的病理生理意义。由于丰富的侧支网络，胰腺主要动脉闭塞不会引起病理变化。静脉流出完全闭塞会导致胰腺出血性坏死、微循环紊乱、水肿以及如急性胰腺炎中的局灶性实质坏死。在最后一种模型中，可以检查胰腺和全身的改变。
