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关于缺血性心肌损伤的机制

On the mechanisms of ischaemic myocardial lesions.

作者信息

Jung J

出版信息

Morphol Embryol (Bucur). 1979 Oct-Dec;25(4):323-8.

PMID:160987
Abstract

The examination of the myocardium in 609 natural and violent death cases by using Lie's histochemical method revealed recent ischaemic lesions, extended or microfocal, in 511 cases. These lesions were prevailingly extended in sudden cardiac deaths, alcoholic intoxication and electrocution, while in the various kinds of shock, in asphyxia and pesticide poisoning cases they were of a predominantly microfocal nature. According to these morphological changes, the vascular and metabolic factors involved in producing ischaemic cardiomyopathy are discussed.

摘要

运用李氏组织化学方法对609例自然死亡和暴力死亡病例的心肌进行检查,结果显示511例存在近期缺血性病变,病变呈扩展性或微灶性。这些病变在心脏猝死、酒精中毒和触电死亡病例中以扩展性为主,而在各类休克、窒息和农药中毒病例中则以微灶性为主。根据这些形态学变化,对导致缺血性心肌病的血管和代谢因素进行了讨论。

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