Matsumoto K, Hukuda S, Nishioka J, Asajima S
Department of Orthopaedic Surgery, Shiga University of Medical Science, Japan.
Clin Orthop Relat Res. 1992 Jul(280):235-40.
Spontaneous Achilles tendon rupture occurred in a 60-year-old man with a two-year history of sero-positive rheumatoid arthritis (RA). Histologic section of the ruptured Achilles tendon revealed the existence of rheumatoid granulation that consisted of lymphocytes, histiocytes, and small vascular proliferation within the tendon tissue. The enthesis of the tendon to the calcaneus also revealed lymphocyte infiltration in the bone marrow and rheumatoid granulation within the tendon. Throughout the course of his disease, the patient had not been treated with corticosteroids. These facts suggested that enthesis (attachment of tendon to bone) was one of the extraarticular sites of the rheumatoid inflammation and that enthesitis was a possible cause of Achilles tendon rupture in this RA patient.
一名60岁男性，有两年血清阳性类风湿关节炎（RA）病史，发生了自发性跟腱断裂。对断裂的跟腱进行组织学切片检查发现存在类风湿肉芽组织，其由肌腱组织内的淋巴细胞、组织细胞和小血管增生组成。肌腱与跟骨的附着点也显示骨髓中有淋巴细胞浸润以及肌腱内有类风湿肉芽组织。在整个病程中，该患者未接受过皮质类固醇治疗。这些事实表明，附着点（肌腱与骨的附着处）是类风湿炎症的关节外部位之一，并且附着点炎可能是该类风湿关节炎患者跟腱断裂的一个原因。
