Luong Amber, Roland Peter S
Department of Otolaryngology-Head and Neck Surgery, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA.
Curr Opin Otolaryngol Head Neck Surg. 2005 Oct;13(5):273-6. doi: 10.1097/01.moo.0000172821.04946.30.
Although uncommon, acquired external auditory canal stenosis is an entity that is encountered by many otolaryngologists. This paper reviews the causes, pathogenesis, assessment, and management options for external auditory canal stenosis.
Initially, acquired external auditory canal stenosis was described as resulting from a number of different causes. Since then, histology and imaging studies of this disease have shown that a common cascade of inflammatory changes resulting from these different causes is the primary pathogenesis leading to medial canal fibrosis. Once there is complete obstruction of the external auditory canal, surgery is the primary treatment. Understanding the strong role of inflammation in the pathogenesis of external auditory canal stenosis may lead to new preventative medical therapies.
Although acquired external auditory canal was described several years ago, its underlying pathophysiology is now better understood. From this knowledge, improved medical as well as surgical approaches can be developed.
尽管后天性外耳道狭窄并不常见，但许多耳鼻喉科医生都会遇到这种情况。本文综述了后天性外耳道狭窄的病因、发病机制、评估方法及治疗选择。
最初，后天性外耳道狭窄被描述为由多种不同原因引起。从那时起，对该疾病的组织学和影像学研究表明，这些不同原因导致的一系列常见炎症变化是导致外耳道内侧纤维化的主要发病机制。一旦外耳道完全阻塞，手术是主要治疗方法。了解炎症在外耳道狭窄发病机制中的重要作用可能会带来新的预防性医学疗法。
尽管后天性外耳道狭窄在几年前就已被描述，但现在对其潜在的病理生理学有了更好的理解。基于这一认识，可以开发出更好的医学和手术治疗方法。
