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[Carbapenem resistance in Pseudomonas aeruginosa isolates: an example of interaction between different mechanisms].
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The relative contribution of efflux and target gene mutations to fluoroquinolone resistance in recent clinical isolates of Pseudomonas aeruginosa.
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Multidrug resistance associated with mexXY expression in clinical isolates of Pseudomonas aeruginosa from a Texas hospital.
Diagn Microbiol Infect Dis. 2004 Sep;50(1):43-50. doi: 10.1016/j.diagmicrobio.2004.05.004.
2
Insertional inactivation of oprD in clinical isolates of Pseudomonas aeruginosa leading to carbapenem resistance.
FEMS Microbiol Lett. 2004 Jul 1;236(1):137-43. doi: 10.1016/j.femsle.2004.05.039.
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Hypersusceptibility of the Pseudomonas aeruginosa nfxB mutant to beta-lactams due to reduced expression of the ampC beta-lactamase.
Antimicrob Agents Chemother. 2001 Apr;45(4):1284-6. doi: 10.1128/AAC.45.4.1284-1286.2001.
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Complete genome sequence of Pseudomonas aeruginosa PAO1, an opportunistic pathogen.
Nature. 2000 Aug 31;406(6799):959-64. doi: 10.1038/35023079.
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Characterization of MexT, the regulator of the MexE-MexF-OprN multidrug efflux system of Pseudomonas aeruginosa.
J Bacteriol. 1999 Oct;181(20):6300-5. doi: 10.1128/JB.181.20.6300-6305.1999.
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Carbapenem activities against Pseudomonas aeruginosa: respective contributions of OprD and efflux systems.
Antimicrob Agents Chemother. 1999 Feb;43(2):424-7. doi: 10.1128/AAC.43.2.424.
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Characterization of the MexC-MexD-OprJ multidrug efflux system in DeltamexA-mexB-oprM mutants of Pseudomonas aeruginosa.
Antimicrob Agents Chemother. 1998 Aug;42(8):1938-43. doi: 10.1128/AAC.42.8.1938.
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Susceptibility to beta-lactam antibiotics of Pseudomonas aeruginosa overproducing penicillin-binding protein 3.
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