Fitzgerald Mark, Spencer Jack, Johnson Fiona, Marasco Silvana, Atkin Chris, Kossmann Thomas
National Trauma Research Institute, The Alfred, Melbourne, Victoria, Australia.
Emerg Med Australas. 2005 Oct-Dec;17(5-6):494-9. doi: 10.1111/j.1742-6723.2005.00782.x.
Blunt cardiac injuries are a leading cause of fatalities following motor-vehicle accidents. Injury to the heart is involved in 20% of road traffic deaths. Structural cardiac injuries (i.e. chamber rupture or perforation) carry a high mortality rate and patients rarely survive long enough to reach hospital. Chamber rupture is present at autopsy in 36-65% of death from blunt cardiac trauma, whereas in clinical series it is present in 0.3-0.9% of cases and is an uncommon clinical finding. Patients with large ruptures or perforations usually die at the scene or in transit--the rupture of a cardiac cavity, coronary artery or intrapericardial portion of a major vein or artery is usually instantly fatal because of acute tamponade. The small, rare, remaining group of patients who survive to hospital presentation usually have tears in a cavity under low pressure and prompt diagnosis and surgery can now lead to a survival rate of 70-80% in experienced trauma centres. As regional trauma systems evolve, patients with severe, but potentially survivable cardiac injury are surviving to ED. Two distinct syndromes are apparent--haemorrhagic shock and cardiac tamponade. Any patient with severe chest trauma, hypotension disproportionate to estimated loss of blood or with an inadequate response to fluid administration should be suspected of having a cardiac cause of shock. For patients with severe hypotension or in extremis, the treatment of choice is resuscitative thoracotomy with pericardotomy. Closed chest cardiopulmonary resuscitation is ineffective in these circumstances. Blunt traumatic cardiac injury presenting with shock is associated with a poor prognosis. The majority of survivors of blunt or penetrating cardiac injury present to the ED/trauma centre with vital signs. The main pathophysiologic determinant for most survivors is acute pericardial tamponade. The presence of normal clinical signs or normal ECG studies does not exclude tamponade. In recent years the widespread availability and use of ultrasound for the initial assessment of severely injured patients has facilitated the early diagnosis of cardiac tamponade and associated cardiac injuries. Two cases of survival from blunt traumatic cardiac trauma are described in the present paper to demonstrate survivability in the context of rapid assessment and intervention.
钝性心脏损伤是机动车事故后死亡的主要原因。心脏损伤占道路交通死亡人数的20%。心脏结构损伤(即心室破裂或穿孔)死亡率很高,患者很少能存活到医院。在钝性心脏创伤死亡的尸检中,36% - 65%存在心室破裂,而在临床系列中,仅0.3% - 0.9%的病例存在心室破裂,这是一种不常见的临床发现。大的破裂或穿孔患者通常在现场或转运途中死亡——心脏腔室、冠状动脉或主要静脉或动脉的心包内部分破裂通常因急性心包填塞而立即致命。少数能存活到医院就诊的患者通常是低压腔室有撕裂伤,在经验丰富的创伤中心,及时诊断和手术现在可使存活率达到70% - 80%。随着区域创伤系统的发展,严重但有可能存活的心脏损伤患者能存活到急诊科。有两种不同的综合征很明显——失血性休克和心包填塞。任何有严重胸部创伤、低血压与估计失血量不成比例或对液体输注反应不佳的患者都应怀疑有心脏性休克的原因。对于严重低血压或处于危急状态的患者,首选治疗方法是开胸复苏并心包切开术。在这些情况下,胸外心脏按压无效。钝性创伤性心脏损伤伴休克的预后很差。大多数钝性或穿透性心脏损伤的幸存者在急诊科/创伤中心就诊时生命体征尚正常。大多数幸存者的主要病理生理决定因素是急性心包填塞。正常的临床体征或正常的心电图检查并不能排除心包填塞。近年来,超声在重伤患者初始评估中的广泛应用和使用有助于早期诊断心包填塞及相关心脏损伤。本文描述了两例钝性创伤性心脏创伤存活的病例,以证明在快速评估和干预情况下的可存活情况。