Gyhra A, Pierart J, Torres P, Prieto L
Department of Surgery, University of Concepción, Chile.
J Trauma. 1992 Jul;33(1):25-8.
In order to demonstrate that the evolution of cardiac tamponade from a ventricular wound is different from that without myocardial wounding, the effects of a rapid infusion of saline solution on hemodynamic behavior and pericardial pressure (PP) were evaluated in dogs with cardiac tamponade caused by ventricular perforation (group C), animals without cardiac tamponade (group A), and animals with cardiac tamponade induced by infusion of saline into the pericardium (group B). We found that blood pressure (BP) increased from 107.5 +/- 15.5 mm Hg to 126 +/- 4 mm Hg in group A; increased from 64.5 +/- 17.9 mm Hg to 117.5 +/- 22.17 mm Hg in group B; and increased from 60.75 +/- 46.5 mm Hg to 76 +/- 14.4 mm Hg in group C. Central venous pressure (CVP) increased from 3.75 +/- 0.96 cm H2O to 9.5 +/- 3.3 cm H2O in group A; increased from 8 +/- 2.4 cm H2O to 16.25 +/- 3.1 cm H2O in group B; and rose from 7.75 +/- 2.6 cm H2O to 20.66 +/- 5.03 cm H2O in group C. Cardiac output (CO) increased from 3.9 +/- 1.2 L/min to 18.93 +/- 3.96 L/min in group A; increased from 1.23 +/- 0.3 L/min to 5.4 +/- 1.7 L/min in group B; and increased from 1.8 +/- 0.66 L/min to 3.53 +/- 1.31 L/min in group C.(ABSTRACT TRUNCATED AT 250 WORDS)
为了证明心室创伤导致的心包填塞演变过程与无心肌损伤时不同，我们评估了快速输注盐溶液对因心室穿孔导致心包填塞的犬（C组）、无心包填塞的动物（A组）以及通过向心包内输注盐水诱导心包填塞的动物（B组）血流动力学行为和心包压力（PP）的影响。我们发现，A组血压（BP）从107.5±15.5毫米汞柱升至126±4毫米汞柱；B组从64.5±17.9毫米汞柱升至117.5±22.17毫米汞柱；C组从60.75±46.5毫米汞柱升至76±14.4毫米汞柱。A组中心静脉压（CVP）从3.75±0.96厘米水柱升至9.5±3.3厘米水柱；B组从8±2.4厘米水柱升至16.25±3.1厘米水柱；C组从7.75±2.6厘米水柱升至20.66±5.03厘米水柱。A组心输出量（CO）从3.9±1.2升/分钟增至18.93±3.96升/分钟；B组从1.23±0.3升/分钟增至5.4±1.7升/分钟；C组从1.8±0.66升/分钟增至3.53±1.31升/分钟。（摘要截选至250词）
