Bertone Armando, Faubert Jocelyn
Visual Psychophysics and Perception Laboratory, Ecole d'optométrie, Université de Montréal, Canada.
J Autism Dev Disord. 2006 Jan;36(1):55-64. doi: 10.1007/s10803-005-0042-5.
Interest regarding neural information processing in autism is growing because atypical perceptual abilities are a characteristic feature of persons with autism. Central to our review is how characteristic perceptual abilities, referred to as perceptual signatures, can be used to suggest a neural etiology that is specific to autism. We review evidence from studies assessing both motion and form perception and how the resulting perceptual signatures are interpreted within the context of two main hypotheses regarding information processing in autism: the pathway- and complexity-specific hypotheses. We present evidence suggesting that an autism-specific neural etiology based on perceptual abilities can only be made when particular experimental paradigms are used, and that such an etiology is most congruent with the complexity-specific hypothesis.
由于非典型感知能力是自闭症患者的一个特征，因此人们对自闭症中神经信息处理的兴趣与日俱增。我们综述的核心是，如何利用被称为感知特征的特征性感知能力来推断自闭症特有的神经病因。我们回顾了评估运动感知和形状感知的研究证据，以及如何在关于自闭症信息处理的两个主要假设（通路特异性假设和复杂性特异性假设）的背景下解释由此产生的感知特征。我们提供的证据表明，只有使用特定的实验范式，才能基于感知能力得出自闭症特有的神经病因，而且这种病因与复杂性特异性假设最为一致。
