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Nongenetic complementation of group V temperature-sensitive mutants of vesicular stomatitis virus by UV-irradiated virus.紫外线照射病毒对水疱性口炎病毒V组温度敏感突变体的非遗传互补作用
J Virol. 1975 Apr;15(4):798-805. doi: 10.1128/JVI.15.4.798-805.1975.
2
Nongenetic complementation in VSV: asymmetric contribution of the L proteins of each parent in the rescue of group I ts mutants.水泡性口炎病毒中的非遗传互补：每个亲本的L蛋白在I组温度敏感突变体拯救中的不对称贡献。
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Temperature-sensitive mutant TS O82 of vesicular stomatitis virus. I. Rescue at nonpermissive temperature by uv-irradiated virus.
Virology. 1979 Feb;93(1):286-90. doi: 10.1016/0042-6822(79)90301-5.
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Synthesis of RNA by mutants of vesicular stomatitis virus (Indiana serotype) and the ability of wild-type VSV New Jersey to complement the VSV Indiana ts G I-114 transcription defect.水泡性口炎病毒（印第安纳血清型）突变体对RNA的合成以及野生型VSV新泽西株对VSV印第安纳ts G I-114转录缺陷的互补能力。
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Parental G protein reincorporation by a vesicular stomatitis virus temperature-sensitive mutant of complementation group V at nonpermissive temperature.在非允许温度下，水泡性口炎病毒互补组V的温度敏感突变体使亲本G蛋白重新掺入。
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Rescue at nonpermissive temperature of complementation group II temperature-sensitive mutants of vesicular stomatitis virus by uv-irradiated VSV.
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Primary in vivo transcription of vesicular stomatitis virus and temperature-sensitive mutants of five vesicular stomatitis virus complementation groups.水泡性口炎病毒及其五个互补组的温度敏感突变体的体内初级转录
J Virol. 1973 Dec;12(6):1238-52. doi: 10.1128/JVI.12.6.1238-1252.1973.

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1
UV irradiation analysis of complementation between, and replication of, RNA-negative temperature-sensitive mutants of Newcastle disease virus.新城疫病毒RNA阴性温度敏感突变体之间的互补及复制的紫外线照射分析
J Virol. 1982 Mar;41(3):965-73. doi: 10.1128/JVI.41.3.965-973.1982.
2
The genetics of vesiculoviruses.水泡性病毒的遗传学
Arch Virol. 1982;72(1-2):1-34. doi: 10.1007/BF01314447.

本文引用的文献

1
The effects of ultraviolet irradiation on the transcription of T4 DNA.紫外线照射对T4 DNA转录的影响。
Biochim Biophys Acta. 1970;209(2):368-86. doi: 10.1016/0005-2787(70)90735-5.
2
Bacteriophage f1 infection: fate of the parental major coat protein.噬菌体f1感染：亲本主要外壳蛋白的命运
J Virol. 1974 Jan;13(1):94-9. doi: 10.1128/JVI.13.1.94-99.1974.
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Isolation and characterization of temperature-sensitive mutants of vesicular stomatitis virus, New Jersey serotype.水泡性口炎病毒新泽西血清型温度敏感突变体的分离与鉴定
J Virol. 1971 Dec;8(6):836-41. doi: 10.1128/JVI.8.6.836-841.1971.
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Inhibitor of vesicular stomatitis virus transcriptase in purified virions.
Nature. 1974 Mar 1;248(5443):45-7. doi: 10.1038/248045a0.
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Temperature-sensitive mutants of vesicular stomatitis virus; homology and nomenclature.水泡性口炎病毒的温度敏感突变体；同源性与命名
J Gen Virol. 1973 Jun;19(3):295-300. doi: 10.1099/0022-1317-19-3-295.
6
A search for recombination between temperature-sensitive mutants of vesicular stomatitis virus.对水泡性口炎病毒温度敏感突变体之间重组的研究。
J Gen Virol. 1971 Dec;13(3):477-9. doi: 10.1099/0022-1317-13-3-477.
7
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.水泡性口炎病毒感染的L细胞中蛋白质合成的抑制作用
J Virol. 1972 Jan;9(1):85-9. doi: 10.1128/JVI.9.1.85-89.1972.
8
Relationship between the ribonucleic acid synthesizing capacity of ultraviolet-irradiated Newcastle disease virus and its ability to induce interferon.紫外线照射的新城疫病毒的核糖核酸合成能力与其诱导干扰素的能力之间的关系
J Virol. 1971 Oct;8(4):500-8. doi: 10.1128/JVI.8.4.500-508.1971.
9
[Analysis of a thermolabile mutant of vesicular stomatitis virus (VSV)].
Ann Inst Pasteur (Paris). 1971 Jul;121(1):101-6.
10
The homologies of spontaneous and induced temperature-sensitive mutants of vesicular stomatitis virus isolated in chick embryo and BHK 21 cells.在鸡胚和BHK 21细胞中分离的水泡性口炎病毒自发和诱导的温度敏感突变体的同源性。
J Gen Virol. 1971 May;11(2):81-5. doi: 10.1099/0022-1317-11-2-81.

紫外线照射病毒对水疱性口炎病毒V组温度敏感突变体的非遗传互补作用

Nongenetic complementation of group V temperature-sensitive mutants of vesicular stomatitis virus by UV-irradiated virus.

作者信息

Deutsch V

出版信息

J Virol. 1975 Apr;15(4):798-805. doi: 10.1128/JVI.15.4.798-805.1975.

DOI:10.1128/JVI.15.4.798-805.1975
PMID:163919
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC354522/
Abstract

Cells infected with temperature-sensitive (ts) mutants of complementation group V of vesicular stomatitis virus (VSV) give an enhanced yield at nonpermissive temperature when co-infected or superinfected with UV-irradiated virus. Virions produced in these mixed infections are temperature sensitive and do not complement ts V45. Rescue of group V mutants is multiplicity dependent. It can occur in the presence of cycloheximide; kinetics of rescue are similar in the absence or in the presence of the drug. Rescue is due to nongenetic complementation and is interpreted as a trigger effect on maturation of a small quantity of biologically active protein V molecules provided by UV-irradiated virus. These results are comfirmed by rescue of ts V45 by UV-irradiated, defective, interferring T particles.

摘要

感染水疱性口炎病毒（VSV）互补组V温度敏感（ts）突变体的细胞，在与紫外线照射的病毒共感染或超感染时，在非允许温度下产量会提高。在这些混合感染中产生的病毒粒子对温度敏感，并且不能互补ts V45。V组突变体的拯救是多倍体依赖性的。它可以在环己酰亚胺存在的情况下发生；在药物不存在或存在的情况下，拯救的动力学相似。拯救是由于非遗传互补，并且被解释为对紫外线照射的病毒提供的少量生物活性蛋白V分子成熟的触发作用。这些结果通过紫外线照射的、有缺陷的、干扰性T颗粒对ts V45的拯救得到证实。