Rende Richard, Slomkowski Cheryl, Lloyd-Richardson Elizabeth, Niaura Raymond
Centers for Behavioral & Preventive Medicine, Brown Medical School, Providence, RI 02906, USA.
J Fam Psychol. 2005 Dec;19(4):611-8. doi: 10.1037/0893-3200.19.4.611.
Prior research on sibling contagion for substance use has not attended to individual differences in the sibling relationship that may be influenced by genetic similarity. The authors utilizing data on a sample of twin and nontwin siblings participating in the National Longitudinal Study of Adolescent Health (Add Health). Although monozygotic twins had the highest levels of sibling contact and mutual friendships, the pattern of results for other sibling types were not consistent with genetic models, and biometric analysis indicated that shared environmental factors influenced these sibling relationship features. Application of DeFries-Fulker regression models provided evidence that sibling contact and mutual friendships represent a source of social contagion for adolescent smoking and drinking independent of genetic relatedness. The results are interpreted using a social contagion framework and contrasted with other competing models such as those focused on the equal environments assumption and niche selection.
先前关于物质使用的同胞传染的研究没有关注到同胞关系中的个体差异,而这种差异可能受到基因相似性的影响。作者利用了参与青少年健康全国纵向研究(Add Health)的双胞胎和非双胞胎同胞样本的数据。尽管同卵双胞胎的同胞接触和相互友谊程度最高,但其他同胞类型的结果模式与基因模型不一致,生物统计学分析表明,共享环境因素影响了这些同胞关系特征。DeFries-Fulker回归模型的应用提供了证据,表明同胞接触和相互友谊是青少年吸烟和饮酒的社会传染源,与基因相关性无关。研究结果使用社会传染框架进行解释,并与其他竞争模型进行对比,如那些侧重于平等环境假设和生态位选择的模型。